机译:AP-2α基因敲除小鼠表现出视杯图案缺陷和视杆形态发生失败
Department of Pathology and Molecular Medicine, McMaster University, Hamilton, ON, Canada,;
Department of Craniofacial Biology and|Department of Cell and Developmental Biology, University of Colorado Denver, Anschutz Medical Campus, Aurora, CO, USA,;
Department of Ophthalmology and Visual Sciences,|Department of Cell and Developmental Biology and;
Department of Ophthalmology and Visual Sciences,|Department of Cell and Developmental Biology and|Department of Cellular and Molecular Biology, University of Michigan Medical School, Ann Arbor, MI, USA and;
Department of Ophthalmology and Visual Sciences, Moran Eye Center, University of Utah, Salt Lake City, UT, USA;
机译:AP-2alpha基因敲除小鼠表现出视杯图案缺陷和视杆形态发生失败。
机译:眼外胚层β-catenin功能获得性小鼠的视杯和面部图案缺陷
机译:视网膜中活化蛋白2α(AP-2α)的条件删除表明AP-2α在视杯发育中的非细胞自主作用
机译:基于OPTICS和多标签分类的晶圆缺陷模式识别
机译:视杯形态发生和神经视网膜发育中转录因子AP-2的细胞自主和非细胞自主要求
机译:AP-2α基因敲除小鼠表现出视杯图案缺陷和视杆形态发生失败
机译:AP-2α基因敲除小鼠表现出视杯图案缺陷和视杆形态发生失败