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机译:caspase-6缺陷小鼠的兴奋性毒性和轴突变性的挽救,以及年龄依赖性的行为和神经解剖学改变
1Department of Medical Genetics, Centre for Molecular Medicine and Therapeutics, Child and Family Research Institute, University of British Columbia, Vancouver, British Columbia V5Z 4H4, Canada, 2Department of Cell and Developmental Biology and Neuroscience Center, University of North Carolina, Chapel Hill, NC 27599-7250, USA, 3Department of Medical Genetics, Institute of Cellular and Molecular Medicine, University of Copenhagen, 2200 N Copenhagen, Denmark, 4Developmental and Stem Cell Biology Group, Hospital for Sick Children, Toronto, Ontario M5G1L7, Canada, 5Department of Molecular Genetics and 6Department of Physiology, University of Toronto, Toronto, Ontario M5G1X5, Canada, 7CHDI Foundation, Inc., New York, NY 10001, USA and 8Taconic Biosciences, Cranbury, NJ 08512, USA;
机译:caspase-6缺陷小鼠的兴奋性毒性和轴突变性的挽救,以及年龄依赖性的行为和神经解剖学改变
机译:spock3突变小鼠的call体和皮质轴突结构异常,伴随着焦虑样行为的减少和社交能力的降低。
机译:在小鼠病毒疾病中神经元,突触和轴突病理学的中心:与丘脑和脑干核相关的神经解剖学联系
机译:长时间经角膜电刺激后视网膜变性小鼠的脑电图功率变化
机译:抑制小鼠14-3-3蛋白会导致精神分裂症相关的行为和神经解剖学变化。
机译:caspase-6缺陷小鼠的兴奋性毒性和轴突变性的挽救以及年龄依赖性的行为和神经解剖学改变
机译:减少的人突变体P301S Tau转基因小鼠的轴突运输和增加的兴奋性视网膜神经节细胞变性。