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机译:谷氨酸脱羧酶65(GAD65)基因敲除NOD小鼠自身免疫性糖尿病的发展
Division of Stem Cell Regulation Research G6 Osaka University Graduate School of MedicineDepartment of Metabolism and Endocrinology Osaka City General Hospital;
Division of Stem Cell Regulation Research G6 Osaka University Graduate School of Medicine;
Division of Stem Cell Regulation Research G6 Osaka University Graduate School of Medicine;
Department of Metabolism and Endocrinology Osaka City General Hospital;
Department of Geriatric Medicine Osaka University Graduate School of Medicine;
Department of Geriatric Medicine Osaka University Graduate School of Medicine;
Department of Biochemistry Institute of Development Aging and Cancer Tohoku University;
Laboratory of Neurochemistry National Institute for Physiological Sciences;
Division of Stem Cell Regulation Research G6 Osaka University Graduate School of Medicine;
GAD65; autoantigen; autoimmune diabetes; knockout mouse; NOD mouse;
机译:致耐受性DC(tolDC)的抗原负荷(例如谷氨酸脱羧酶65)降低了其预防非肥胖糖尿病(NOD)-糖尿病和NOD小鼠过继共转移的严重联合免疫功能低下模型的能力。
机译:利用生物素-GAD65融合蛋白开发谷氨酸脱羧酶65(GAD65)自身抗体的方法
机译:日本非胰岛素依赖型糖尿病患者的谷氨酸脱羧酶65(GAD65)抗体和胰岛素自身抗体
机译:预防IL-4和IL-10质粒递送的NOD小鼠自身免疫性糖尿病。
机译:MHC II类分子在I型糖尿病中的作用:与糖尿病相关的HLA-DQ8分子对谷氨酸脱羧酶65表达的分析。
机译:使用DQ8四聚体评估CD4 + T细胞对谷氨酸脱羧酶65的反应揭示了GAD65 121–140和GAD65 250–266在T1D发育中的致病作用
机译:谷氨酸脱羧酶65(GAD65)敲除Nod小鼠的自身免疫糖尿病