机译：脂联素通过抑制p38 MAP激酶/ p16 ^ sup INK4A ^信号通路来预防糖尿病祖细胞的过早衰老并促进内皮修复。
Junlei Chang,1,2 Timing Ii,3 Yu Huang,4 Karen S.L. Lam,1,2 Ruby L.C. Hoo,1,2 Wing Tak Wong,4 Kenneth K.Y. Cheng,1,2 Yiqun Wang,1,2 Paul M. Vanhoutte,2,8 and Aimin Xu1,2,5From the 1 Department of Medicine, University of Hong Kong, Hong Kong, China, the 2 Research Center for Heart, Brain, Hormones, and Healthy Aging, Univeristy of Hong Kong, Hong Kong, China, the 3 Department of Endocrinology, Huashan Hospital, Fudan University, Shanghai, China, the 4 Institute of Vascular Medicine, Li Ka Shing Institute of Health Sciences and School of Biomédical Sciences, Chinese University of Hong Kong, Hong Kong, China, and the Department of Pharmacology and Pharmacy, University of Hong Kong, Hong Kong, China.Corresponding author Aimin Xu, amxu@hkucc,hku.hk.Received 25 April 2010 and accepted 20 August 2010. Published ahead of print at httprfdiabetes.diabetesuournals.org on 29 August 2010. DOI: 10.2337/ db10-0582.© 2010 by the American Diabetes Association. Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered. See http://creauvecommons.org/licenses/by -nc-nd/3.0/ for details.The casts of publication o/ (AM article were defrayed in part by the payment of page charges. This article must therefore be hereby marked "advertisement" in accordance with 18 U.S.C. Section 1734 solely to indicate this fact.,;
机译：脂联素通过抑制p38 MAP激酶/ p16〜（INK4A）信号通路防止糖尿病祖细胞内皮细胞过早衰老并促进内皮细胞修复
机译：细胞周期蛋白依赖性激酶抑制剂p16（INK4a）和端粒酶可能共调节内皮祖细胞衰老。
机译：银杏内酯B促进骨髓源性内皮祖细胞的增殖和功能活性：Akt / eNOS和MAPK / p38信号通路的参与
机译：脂多糖（LPS）诱导人体内皮细胞（EC）中的信号传导。 G蛋白的作用和P38映射激酶途径
机译：在体外和体内，p38 MAPK介导从心肌细胞向内皮细胞旁分泌信号传导的作用。
机译：勘误表。脂联素通过抑制p38 MAP激酶/ p16INK4A信号通路来预防糖尿病祖细胞的过早衰老并促进内皮修复。糖尿病2010； 59：2949–2959
机译：脂联素通过抑制p38 MAP激酶/ p16INK4A信号通路来预防糖尿病祖细胞的过早衰老并促进内皮修复
