机译:肝糖原超补偿激活AMP激活的蛋白激酶,削弱胰岛素信号传导,并减少肝脏中糖原的沉积
Jason J. Winnick,1 Zhibo An,1 Christopher J. Ramnanan,1 Marta Smith,1 Jose M. Irimia,2 Doss W. Neal,1 Mary Courtney Moore,1 Peter J. Roach,2 and Alan D. Cherrington1From the 1Department of Molecular Physiology and Biophysics, Vanderbilt University Medical Center, Nashville, Tennessee, and the "Department of Biochemistry and Molecular Biology, Indiana University School of Medicine, Indianapolis, Indiana.Corresponding author: Jason J. Winnick, jasoii.vvmnick@vandeibilt.edu.Received 27 April 2010 and accepted 29 October 2010.DOI: 10.2337/db 10-0592© 2011 by the American Diabetes Association. Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered. See http://creativeconmions.org/licenses/by -nc-nd/3.0/ for details.,;
机译:肝糖原超补偿激活AMP激活的蛋白激酶,削弱胰岛素信号传导,并减少肝脏中糖原的沉积
机译:AMP激活的蛋白激酶磷酸化并灭活肝TI AMP激活的蛋白激酶磷酸化并灭活肝糖原合酶
机译:黄体生成激素通过独立于AKT和丝裂原活化蛋白激酶的途径刺激牛黄体细胞中雷帕霉素信号的哺乳动物靶标:糖原合酶激酶3和AMP活化蛋白激酶的调节。
机译:热应激对热应激奶牛血清胰岛素,脂肪因子,AMP活化蛋白激酶,热休克信号分子的影响以及生物标志物的研究
机译:小鼠长期蛋白质磷酸酶2A抑制作用是否会异常调节外周葡萄糖稳态,降低肝糖原含量并损害肝脏中胰岛素介导的信号转导?
机译:肝糖原超补偿激活AMP激活的蛋白激酶削弱胰岛素信号传导并减少肝脏中糖原的沉积
机译:肝糖原超补偿激活AMP激活的蛋白激酶,削弱胰岛素信号传导,并减少肝脏中糖原的沉积