机译:肝细胞生长因子/ c-Met信号的破坏增强胰腺β细胞死亡并加速糖尿病的发作
Jose Mellado-Gil,1 Taylor C. Rosa,1 Cem Demirci,2 Jose A. Gonzalez-Pertusa,1 Silvia Velazquez-Garcia,1 Sara Ernst,1 Shelley Valle,1 Rupangi C. Vasavada,1 Andrew F. Stewart,1 Laura C. Alonso,1 and Adolfo Garcia-Ocaña1From the 1 Department of Medicine, Division of Endocrinology and Metabolism, University of Pittsburgh, Pittsburgh, Pennsylvania, and the 2 Department of Pediatrics, University of Pittsburgh, Pittsburgh. Pennsylvania.Corresponding author: Adolfo Garcia-Ocaña, ocana@pitt.edu.Received 1 September 2009 and accepted 21 October 2010.DOI: 10.2337/db09-1305This article contains Supplementary Data online at http://diabetes. diabc-lesjournals.org/lookup/suppl/doi:10.2337/db09-130.5/-/DCl.© 2011 by the American Diabetes Association. Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and lhe work is not altered. See http://creativecommons.org/licenses/by -ne-nd/3.0/ for details.,;
机译:肝细胞生长因子/ c-Met信号的破坏增强胰腺β细胞死亡并加速糖尿病的发作
机译:肿瘤基质细胞中的低氧环境通过旁分泌肝细胞生长因子/ c-Met信号传导的激活来加速胰腺癌的进展。
机译:肿瘤基质细胞的低氧环境通过旁分泌肝细胞生长因子/ c-Met信号的激活加速胰腺癌的进展。
机译:C-JUM N-末端激酶参与在人胰腺癌细胞中转导的散发性生长因子信号
机译:表皮生长因子受体在乳腺上皮细胞中肝细胞生长因子信号传导中的作用。
机译:肝细胞生长因子/ c-Met信号的破坏增强胰腺β细胞死亡并加速糖尿病的发作
机译:肝细胞生长因子/ c-Met信号的破坏增强胰腺β细胞死亡并加速糖尿病的发作