机译:TDA651的丢失会通过调节脂肪形成而导致成熟期肥胖,肝脂肪变性和胰岛素抵抗。
Division of Nephrology, Department of Medicine, McMaster University, Hamilton, Ontario, Canada,Hamilton Centre for Kidney Research, St. Joseph's Healthcare Hamilton, Hamilton, Ontario, Canada;
Hamilton Centre for Kidney Research, St. Joseph's Healthcare Hamilton, Hamilton, Ontario, Canada;
Division of Endocrinology and Metabolism, Department of Medicine, McMaster University, Hamilton, Ontario, Canada;
Division of Endocrinology and Metabolism, Department of Medicine, McMaster University, Hamilton, Ontario, Canada;
Department of Pediatrics, University of Colorado School of Medicine, Aurora, Colorado;
Division of Nephrology, Department of Medicine, McMaster University, Hamilton, Ontario, Canada,Hamilton Centre for Kidney Research, St. Joseph's Healthcare Hamilton, Hamilton, Ontario, Canada;
Division of Endocrinology and Metabolism, Department of Medicine, McMaster University, Hamilton, Ontario, Canada;
Department of Pediatrics, University of Colorado School of Medicine, Aurora, Colorado;
Division of Endocrinology and Metabolism, Department of Medicine, McMaster University, Hamilton, Ontario, Canada;
Division of Nephrology, Department of Medicine, McMaster University, Hamilton, Ontario, Canada,Hamilton Centre for Kidney Research, St. Joseph's Healthcare Hamilton, Hamilton, Ontario, Canada;
机译:Biochanin A通过调节饮食诱发的肥胖小鼠的肝脂质和葡萄糖代谢途径来改善肝脂肪变性和胰岛素抵抗
机译:蛋白激酶Cbeta功能的丧失可保护小鼠免受饮食引起的肥胖症以及肝脂肪变性和胰岛素抵抗的发展。
机译:肝癌抑制素M受体β调节肥胖诱导的脂肪变性和胰岛素抵抗
机译:肥胖患者及与肥胖相关的胰岛素抵抗患者的FT-IR唾液分析
机译:肝脏中高脂肪饮食介导的P38α活化有助于胰岛素抵抗和肝脏脂肪变性的发作
机译:TDAG51的缺失导致肥胖肝脂肪变性和胰岛素抵抗通过调节脂肪生成
机译:TDAG51的丧失导致成熟的肥胖,肝脏脂肪变性和通过调节脂肪生成的胰岛素抵抗力