机译:Parp抑制可防止十一个转位酶激活和高血糖诱导的DNA去甲基化
William M. Scholl College of Podiatric Medicine, Rosalind Franklin University of Medicine and Science, North Chicago, IL;
Department of Cell Biology and Anatomy, Chicago Medical School, Rosalind Franklin University of Medicine and Science, North Chicago, IL;
William M. Scholl College of Podiatric Medicine, Rosalind Franklin University of Medicine and Science, North Chicago, IL;
William M. Scholl College of Podiatric Medicine, Rosalind Franklin University of Medicine and Science, North Chicago, IL;
William M. Scholl College of Podiatric Medicine, Rosalind Franklin University of Medicine and Science, North Chicago, IL,Department of Cell Biology and Anatomy, Chicago Medical School, Rosalind Franklin University of Medicine and Science, North Chicago, IL;
机译:聚ADP核糖聚合酶活性的抑制可防止伤口愈合过程中高血糖引起的血管生成受损。
机译:由于对DNA上的PARP1而不是PARP1-DNA诱捕的PARP1引起的PARP1-DNA结合增加与PARP1抑制剂的细胞毒性相关
机译:抑制MEF2A通过阻止心脏成纤维细胞中的Akt和TGF-beta 1 / Smad激活来防止高血糖诱导的细胞外基质积聚
机译:抑制白细胞介素-1beta诱导的MEK / ERK途径的激活和NF-B和AP-1的DNA结合:骨关节炎在骨关节炎中的潜在机制
机译:磷酸三磷酸异构酶是在酶催化中利用结合能的机制模型:活化和抑制研究。
机译:Parp抑制可防止十一个转位酶激活和高血糖诱导的DNA去甲基化
机译:PARP抑制可防止十一十六旋流酶活化和高血糖诱导的DNA去甲基化