机译:抗CD44抗体治疗可降低饮食诱导的肥胖小鼠的高血糖症并改善胰岛素抵抗,脂肪炎症和肝脂肪变性
Division of Systems Medicine, Department of Pediatrics, Stanford University School of Medicine, Stanford, CA,Lucile Packard Children's Hospital, Palo Alto, CA;
Division of Biomedical Research Center, Biomedical Laboratory, Kitasato Institute Hospital, Kitasato University, Minato-ku, Tokyo, Japan;
Department of Diagnostic Pathology, Kitasato Institute Hospital, Kitasato University, Minato-ku, Tokyo, Japan;
Diabetes Center, Kitasato Institute Hospital, Kitasato University, Minato-ku, Tokyo, Japan;
Division of Systems Medicine, Department of Pediatrics, Stanford University School of Medicine, Stanford, CA,Lucile Packard Children's Hospital, Palo Alto, CA;
机译:骨髓骨质HMG-COA还原酶决定脂肪组织炎症,胰岛素抵抗和饮食诱导的肥胖小鼠的肝脏脂肪变性
机译:饮食雌雄三醇减轻饮食诱导的肥胖小鼠的肥胖,肝脂肪变性,胰岛素抵抗和炎症。
机译:沙棘叶中的沙棘叶提取物和类黄酮糖苷提取物可改善饮食诱发的肥胖症,肝脂肪变性,胰岛素抵抗和炎症。
机译:要进行对数转换或不进行对数转换:在以腹部肥胖作为炎症和胰岛素抵抗的预测因子的模型中,对数正态数据进行回归
机译:脂蛋白受体SR-B1缺乏增强脂肪组织炎症,并在小鼠饮食诱导的肥胖期间减少对肝脏脂肪变性的易感性
机译:抗CD44抗体治疗可降低饮食诱导的肥胖小鼠的高血糖症并改善胰岛素抵抗脂肪炎症和肝脂肪变性
机译:抗CD44抗体治疗降低高血糖,提高胰岛素抵抗,脂肪炎症和饮食诱导的肥胖小鼠的肝脏脂肪变性