机译:年轻人中ABCA1的功能丧失突变和增强的β细胞分泌能力
Division of Endocrinology, Diabetes, and Metabolism, Department of Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA;
Division of Translational Medicine and Human Genetics, Department of Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA;
Division of Endocrinology, Diabetes, and Metabolism, Department of Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA;
Division of Translational Medicine and Human Genetics, Department of Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA;
Division of Translational Medicine and Human Genetics, Department of Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA;
Department of Biochemistry and Biophysics, Department of Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA;
Robarts Research Institute, University of Western Ontario, London, Ontario, Canada;
Division of Translational Medicine and Human Genetics, Department of Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA;
机译:ABCA1功能丧失突变的携带者表现出胰腺β细胞功能障碍。
机译:成年深色精原细胞与隐睾症的Leydig细胞分泌能力之间的关系。
机译:垂体细胞平移和分泌能力是通过转录因子CREB3L2自治的增强的细胞
机译:首先到达那里:适应性损失突变的进化率优势
机译:参与腺苷受体介导的泡沫细胞形成抑制的分子机制:A2A腺苷受体激活通过增强ABCA1和27-羟化酶依赖性胆固醇外排而减少泡沫细胞形成。
机译:年轻人中ABCA1的功能丧失突变和增强的β细胞分泌能力
机译:ABCA1功能丧失突变的载体显示胰腺β细胞功能异常。