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机译:TCF7L2的糖尿病相关变异与肝或肝外胰岛素抵抗无关
Division of Endocrinology, Diabetes & Metabolism, Mayo Clinic College of Medicine, Rochester, MN;
Center for Neurosciences and Cell Biology, Department of Zoology, University of Coimbra, Coimbra, Portugal;
Center for Neurosciences and Cell Biology, Department of Zoology, University of Coimbra, Coimbra, Portugal;
Center for Neurosciences and Cell Biology, Department of Zoology, University of Coimbra, Coimbra, Portugal;
Division of Endocrinology, Diabetes & Metabolism, Mayo Clinic College of Medicine, Rochester, MN;
Division of Endocrinology, Diabetes & Metabolism, Mayo Clinic College of Medicine, Rochester, MN;
Center for Neurosciences and Cell Biology, Department of Zoology, University of Coimbra, Coimbra, Portugal;
Division of Endocrinology, Diabetes & Metabolism, Mayo Clinic College of Medicine, Rochester, MN;
机译:TCF7L2 rs7903146的遗传变异和GDM的历史对糖尿病相关的代谢性状产生负面影响
机译:TCF7L2中糖尿病相关的遗传变异可以教导我们关于2型糖尿病的发病机制吗?
机译:肝和肝外胰岛素抵抗对空腹血糖受损的发病机制的贡献:糖异生率增加的作用
机译:用综合霰弹枪/靶向定量蛋白质组学策略调查肝胰岛素抵抗
机译:胰岛素受体是肝胰岛素抵抗的控制位点。
机译:TCF7L2的糖尿病相关变异与肝或肝外胰岛素抵抗无关
机译:肝和肝外胰岛素抵抗对空腹血糖受损的发病机制的贡献:糖异生率增加的作用