机译:在1型糖尿病小鼠模型中,抑制12 / 15-脂氧合酶可抵抗β细胞氧化应激和血糖下降。
Department of Pediatrics and the Center for Diabetes and Metabolic Diseases, Indiana University School of Medicine, Indianapolis, IN;
Department of Chemistry, Purdue Institute for Drug Discovery, Purdue Center for Cancer Research,Purdue Institute for Inflammation, Immunology and Infectious Disease,Purdue Institute for Integrative Neuroscience, Purdue University, West Lafayette, IN;
Department of Chemistry, Purdue Institute for Drug Discovery, Purdue Center for Cancer Research,Purdue Institute for Inflammation, Immunology and Infectious Disease,Purdue Institute for Integrative Neuroscience, Purdue University, West Lafayette, IN;
Department of Biochemistry and Molecular Biology, Indiana University School of Medicine, Indianapolis, IN;
Department of Pediatrics and the Center for Diabetes and Metabolic Diseases, Indiana University School of Medicine, Indianapolis, IN,Department of Cellular and Integrative Physiology, Indiana University School of Medicine, Indianapolis, IN;
Department of Pediatrics and the Center for Diabetes and Metabolic Diseases, Indiana University School of Medicine, Indianapolis, IN,Department of Cellular and Integrative Physiology, Indiana University School of Medicine, Indianapolis, IN,Department of Medicine, Eastern Virginia Medical School, Norfolk, VA;
Department of Pediatrics and the Center for Diabetes and Metabolic Diseases, Indiana University School of Medicine, Indianapolis, IN;
Department of Pediatrics and the Center for Diabetes and Metabolic Diseases, Indiana University School of Medicine, Indianapolis, IN;
Department of Pediatrics and the Center for Diabetes and Metabolic Diseases, Indiana University School of Medicine, Indianapolis, IN;
Department of Medicine, Eastern Virginia Medical School, Norfolk, VA;
National Center for Advancing Translational Sciences, National Institutes of Health, Rockville, MD;
Department of Pediatrics and the Center for Diabetes and Metabolic Diseases, Indiana University School of Medicine, Indianapolis, IN;
Department of Pediatrics and the Center for Diabetes and Metabolic Diseases, Indiana University School of Medicine, Indianapolis, IN,Department of Biochemistry and Molecular Biology, Indiana University School of Medicine, Indianapolis, IN,Department of Cellular and Integrative Physiology, Indiana University School of Medicine, Indianapolis, IN,Department of Medicine, Indiana University School of Medicine, Indianapolis, IN;
机译:通过涉及抑制细胞外信号调节激酶磷酸化的细胞型特异性机理,氯氮平保护PC-12细胞免受死亡免受死亡的影响。
机译:12 / 15-Lipoxygenase抑制作用抵消了糖尿病周围神经病变的小鼠和细胞培养模型中的MAPK磷酸化
机译:胆固醇标记的小干扰RNA对1型糖尿病小鼠模型中糖尿病肾病参数靶向12/15-脂脂酶的影响
机译:Nrf-2活性抗氧化防御剂可保护小鼠MLE-12肺上皮细胞的促炎和氧化应激
机译:青年和父母对生活质量和糖尿病患者的看法,因为它与糖尿病型1型糖尿病诊断后的第一年患有血糖控制
机译:在1型糖尿病小鼠模型中抑制12 / 15-脂氧合酶可抵抗β细胞氧化应激和血糖下降
机译:抑制12 / 15-脂氧合酶保护1型糖尿病小鼠模型中的β-细胞氧化应激和血糖劣化