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首页> 外文期刊>Journal of Neural Transplantation and Plasticity: Neural Plasticity >Inhibitory Deficits, Delay Aversion and Preschool AD/HD:Implications for the Dual Pathway Model
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Inhibitory Deficits, Delay Aversion and Preschool AD/HD:Implications for the Dual Pathway Model

机译:抑制缺陷,延迟厌恶和幼儿园AD / HD:对双路模型的影响

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The dual pathway model proposes the existence of separate and neurobiologically distinct cognitive (inhibitory and more general executive dysfunction) and motivational (delay aversion) developmental routes to AD/HD. The study reported in this paper explores the relation between inhibitory deficits and delayaversion and their association with AD/HD in a group of three-year-old children. Children identified as having a pre-school equivalent of AD/HD (N=19) and controls (N=19), matched for gender and IQ, completed a battery of inhibition and delay tasks. Correlational and factor analysis supported a dissociation between inhibitory deficits (go-no-go, set shifting) and delay aversion (choice delay) with delay of gratification cross-loading. Childrenwith AD/HD displayed more inhibitory deficits and were more delay averse than controls. The data support the value of the distinction between motivational and cognitive pathways to AD/HD. Furthermore, the data suggest thatsuch a distinction is apparent relatively early on during development.
机译:双路模型提出了分离和神经生物学上不同的认知(抑制性和更一般的执行功能障碍)和励磁(延迟厌恶)发育途径到AD / HD。本文报告的研究探讨了抑制赤字与延迟的关系及其与一群三岁儿童的AD / HD之间的关系。识别为具有用于性别和IQ的AD / HD(n = 19)和控件(n = 19)的学前学前等效的儿童完成了禁止和延迟任务的电池。相关性和因子分析支持抑制缺陷(GO-NO-GO-GO-GO,SET SHIFTING)和延迟厌厌(选择延迟)之间的解离,延迟满足交叉负荷。儿童ad / hd显示出更多的抑制缺陷,并且比控制更延迟。数据支持对AD / HD的激励和认知途径之间的区别的值。此外,数据表明,在开发期间相对较早地区分是显而易见的。

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