Response to Comment on “Cigarette Smoke-Induced Pulmonary Inflammation Is TLR4/MyD88 and IL-1R1/MyD88 Signaling Dependent”

摘要

We investigated the role of TLR4 and MyD88 signaling in an acute cigarette smoke-induced lung inflammation and found that the heat shock protein 70 is up-regulated and is a likely candidate activating TLR4 leading to lung inflammation ([1][1]). These findings agree with those of Maes et al. ([2][2