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Is Anorexia Nervosa a Disorder of the Self? A Psychological Approach

机译:神经性厌食症是一种自我障碍吗?心理学方法

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The debate concerning the pathogenesis and the maintaining factors of eating disorders, anorexia nervosa in particular, is ongoing especially since therapeutic interventions do not result in satisfactory and enduring rates of remission. This paper presents a model for the pathogenesis of eating disorders, based on the hypothesis of a deficiency in the development of the self. We present the theory in light of new evidence concerning the role of attachment insecurity in the development and maintenance of eating disorders. In particular, we define the self in eating disorders in a comprehensive way by taking into account recent evidence from experimental psychology and neurobiology. The paper considers the development of the self in terms of its synchronic (i.e., experienced in the moment) and diachronic (i.e., experienced as continuous over time) aspects. Both synchronic and diachronic aspects of the self are relevant to the expression of eating disorder symptoms. Further, the maturation of the self is interwoven with the development of attachment functioning from infancy to adolescence. This interplay between these developmental processes of the self and of attachment could be crucial in understanding the pathogenesis of eating disorders. The final part of the paper suggests a neurobiological link between the theory of the self in the eating disorders and the spatiotemporal functioning of the brain. Disturbances in spatiotemporal functioning may represent the neurobiological pathway by which deficiencies in the self is related to attachment functions in individuals with eating disorders.
机译:关于饮食失调的发病机理和维持因素,尤其是神经性厌食症的争论仍在进行中,特别是因为治疗性干预措施无法获得令人满意且持久的缓解率。本文提出了一种饮食失调的发病机理模型,该模型基于自我发展缺乏的假设。我们根据有关附件不安全感在饮食失调的发展和维持中的作用的新证据提出了该理论。特别是,我们考虑到来自实验心理学和神经生物学的最新证据,以一种全面的方式定义了饮食失调中的自我。本文从共时性(即瞬间经历)和历时性(即随着时间的推移经历)方面考虑了自我的发展。自我的共时性和历时性都与饮食失调症状的表达有关。此外,自我的成熟与从婴儿期到青春期的依恋的发展交织在一起。自我和依恋的这些发展过程之间的这种相互作用对于理解饮食失调的发病机制可能至关重要。本文的最后一部分提出了饮食失调中的自我理论与大脑时空功能之间的神经生物学联系。时空功能障碍可能代表神经生物学途径,通过该途径,自我缺陷与进食障碍患者的依恋功能有关。

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