Microcirculation changes following acute and chronic portal hypertension and their response towards bacterial translocation challenge

Ricardo Toma; Ana Liberatore; Jose Menchaca-Diaz; Ivan Koh

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Microcirculation changes following acute and chronic portal hypertension and their response towards bacterial translocation challenge

机译：急性和慢性门脉高压后的微循环变化及其对细菌移位挑战的反应

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《Critical care :》 |2008年第5期|共页
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Ricardo Toma; Ana Liberatore; Jose Menchaca-Diaz; Ivan Koh;
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中图分类护理学;
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1. Acute portal hypertension without liver dysfunction enhances bacterial translocation to the lung [J] . RK Toma, RM Silva, AMA Liberatore, Critical care : . 2005,第Suppla2期

机译：没有肝功能障碍的急性门静脉高压症可增加细菌向肺的移位
2. Von Willebrand factor indicates bacterial translocation, inflammation, and procoagulant imbalance and predicts complications independently of portal hypertension severity [J] . Mandorfer M., Schwabl P., Paternostro R., Alimentary pharmacology & therapeutics. . 2018,第7期

机译：Von Willebrand因子表明细菌易位，炎症和促进剂不平衡，并预测独立于门户高血压严重程度的并发症
3. Bacterial translocation and nonselective β-blockers in portal hypertension: Where we are, what we still need [J] . LaMuraV., ColomboM. Gastroenterology . 2014,第1期

机译：门脉高压中的细菌易位和非选择性β受体阻滞剂：我们在哪里，我们仍然需要什么
4. Bacterial Translocation to Mesenteric Lymph Nodes Increases in Chronic Portal Hypertensive Rats [C] . A. Cruz, M. Losada, M.A. LLamas, European Society for Surgical Research . 2008

机译：对肠系膜淋巴结的细菌易位增加慢性门腺高血压大鼠
5. Regulation of renal sodium transporters in response to acute and chronic hypertension. [D] . Magyar, Clara Ethel. 1999

机译：调节肾钠转运蛋白对急性和慢性高血压的反应。
6. Microcirculation changes following acute and chronic portal hypertension and their response towards bacterial translocation challenge [O] . Ricardo Toma, Ana Liberatore, Jose Menchaca-Diaz, 2008

机译：急性和慢性门脉高压后的微循环变化及其对细菌移位挑战的反应
7. Microcirculation changes following acute and chronic portal hypertension and their response towards bacterial translocation challenge [O] . Toma, Ricardo, Liberatore, Ana, Menchaca-Diaz, Jose, 2008

机译：急性和慢性门脉高压后的微循环变化及其对细菌移位挑战的反应

Microcirculation changes following acute and chronic portal hypertension and their response towards bacterial translocation challenge

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