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首页> 外文期刊>Journal of Medical Case Reports >Pseudoinfarction pattern in a patient with hyperkalemia, diabetic ketoacidosis and normal coronary vessels: a case report
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Pseudoinfarction pattern in a patient with hyperkalemia, diabetic ketoacidosis and normal coronary vessels: a case report

机译:高钾血症,糖尿病性酮症酸中毒和冠状动脉正常的患者的伪梗死类型:一例报告

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Introduction A rare electrocardiographic finding of hyperkalemia is ST segment elevation or the so called 'pseudoinfarction' pattern. It has been suggested that hyperkalemia causes the 'pseudoinfarction' pattern not only through its direct myocardial effects, but also through other mechanisms, such as anoxia, acidosis, and coronary artery spasm. Case presentation A 33-year-old Caucasian woman with insulin-treated diabetes presented with continuous epigastric pain of four hours duration. Her coronary heart disease risk factors apart from diabetes included hypercholesterolemia and smoking. Her initial electrocardiogram revealed ST segment elevation in the anteroseptal leads consistent with anterior myocardial infarction. Blood tests revealed hyperglycemia, hyperkalemia, metabolic acidosis and urine ketones, while a bed-side cardiac echocardiogram showed no segmental wall motion abnormality. We provisionally diagnosed diabetic ketoacidosis that was possibly precipitated by acute myocardial infarction, as there were findings in favor of (epigastric pain, electrocardiogram pattern, presence of 3 coronary heart disease risk factors) and against (young age, normal echocardiogram) the diagnosis of acute myocardial infarction. We performed cardiac angiography in order to exclude an anterior acute myocardial infarction, which could lead to myocardial damage and possible severe complications should there be a delay in treatment. Angiography revealed normal coronary arteries. During the procedure, ST segment elevation in the anteroseptal leads was still present in our patient's electrocardiogram results. Conclusion ST segment elevation is a rare manifestation of hyperkalemia. In our patient, coronary spasm did not contribute to such an electrocardiography finding.
机译:简介高钾血症的罕见心电图发现是ST段抬高或所谓的“伪梗塞”型。已经表明,高钾血症不仅通过其直接的心肌作用,而且通过其他机制,例如缺氧,酸中毒和冠状动脉痉挛,引起“假性梗塞”模式。病例介绍一名接受胰岛素治疗的糖尿病的33岁白人妇女表现为持续四个小时的持续上腹痛。除了糖尿病外,她的冠心病危险因素还包括高胆固醇血症和吸烟。她的最初心电图显示前房间隔导联中ST段抬高与前部心肌梗死相符。血液测试显示高血糖,高钾血症,代谢性酸中毒和尿酮,而床旁心脏超声心动图显示无节段性室壁运动异常。我们临时诊断出糖尿病性酮症酸中毒,可能是由急性心肌梗塞引起的,因为发现有利于(电枢痛,心电图模式,3种冠心病危险因素的存在)而反对(年轻,正常超声心动图)的诊断心肌梗塞。我们进行了心脏血管造影以排除急性急性心肌梗死,如果治疗延迟,可能会导致心肌损伤和严重的并发症。血管造影显示冠状动脉正常。在此过程中,我们患者的心电图结果中仍存在前房间隔导联ST段抬高。结论ST段抬高是高钾血症的罕见表现。在我们的患者中,冠状动脉痉挛并未促成这种心电图检查。

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