Helicobacter pylori infection of AZ-521 cells reveals a type IV secretion defect and VacA-independent CagA phosphorylation

Nicole Tegtmeyer; Steffen Backert

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Helicobacter pylori infection of AZ-521 cells reveals a type IV secretion defect and VacA-independent CagA phosphorylation

机译：AZ-521细胞的幽门螺杆菌感染显示IV型分泌缺陷和不依赖VacA的CagA磷酸化

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《Disease models & mechanisms: DMM》 |2017年第12期|共2页
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Nicole Tegtmeyer; Steffen Backert;
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入库时间 2022-08-18 15:48:04

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1. Response to ‘Helicobacter pylori infection of AZ-521 cells reveals a type IV secretion defect and VacA-independent CagA phosphorylation’ [J] . Masayuki Nakano, Toshiya Hirayama Disease models & mechanisms: DMM . 2017,第12期

机译：对“幽门螺杆菌感染AZ-521细胞的反应表明IV型分泌缺陷和不依赖VacA的CagA磷酸化”
2. Expression of CEACAM1 or CEACAM5 in AZ-521 cells restores the type IV secretion deficiency for translocation of CagA by Helicobacter pylori [J] . Tegtmeyer Nicole, Harrer Aileen, Schmitt Verena, Cellular microbiology . 2019,第1期

机译：AZ-521细胞中CeAcam1或CeAcam5的表达恢复了IV型分泌缺乏术通过幽门螺杆菌转位
3. Helicobacter pylori VacA, acting through receptor protein tyrosine phosphatase α, is crucial for CagA phosphorylation in human duodenum carcinoma cell line AZ-521 [J] . Masayuki Nakano, Kinnosuke Yahiro, Eiki Yamasaki, Disease models & mechanisms: DMM . 2016,第12期

机译：幽门螺杆菌VacA通过受体蛋白酪氨酸磷酸酶α起作用，对人十二指肠癌细胞系AZ-521中的CagA磷酸化至关重要
4. Identification of Helicobacter pylori proteins involved in the cag Type-IV secretion system apparatus via discovery and targeted mass spectrometry approaches [C] . Carrie L. Shaffer, W. Hayes McDonald, Salisha Hill, American Society for Mass Spectrometry Conference on Mass Spectrometry and Allied Topics . 2010

机译：通过发现和靶向质谱方法鉴定CAG型-IV分泌系统装置中涉及的幽门螺杆菌蛋白
5. Helicobacter pylori stimulates gastric cancer cell motility through a combination of CagA-dependent and CagA-independent signaling [D] . Snider, Jared Lee 2008

机译：幽门螺杆菌通过依赖于CagA和独立于CagA的信号传导刺激胃癌细胞的运动
6. Type IV secretion of Helicobacter pylori CagA into oral epithelial cells is prevented by the absence of CEACAM receptor expression [O] . Nicole Tegtmeyer, Tabita Denisia Ghete, Verena Schmitt, 2020

机译：缺乏CEACAM受体的表达可以阻止幽门螺杆菌CagA向口腔上皮细胞的IV型分泌。
7. Response to ‘Helicobacter pylori infection of AZ-521 cells reveals a type IV secretion defect and VacA-independent CagA phosphorylation’ [O] . Masayuki Nakano, Toshiya Hirayama 2017

机译：响应'AZ-521细胞的幽门螺杆菌感染的响应显示IV型分泌缺陷和Vaca无关的Caga磷酸化'

Helicobacter pylori infection of AZ-521 cells reveals a type IV secretion defect and VacA-independent CagA phosphorylation

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