首页> 外文期刊>Scandinavian Journal of Trauma, Resuscitation and Emergency Medicine >Decay in chest compression quality due to fatigue is rare during prolonged advanced life support in a manikin model
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Decay in chest compression quality due to fatigue is rare during prolonged advanced life support in a manikin model

机译:在人体模型中延长长期生命支持期间,由于疲劳导致的胸部按压质量下降很少

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Background The aim of this study was to measure chest compression decay during simulated advanced life support (ALS) in a cardiac arrest manikin model. Methods 19 paramedic teams, each consisting of three paramedics, performed ALS for 12 minutes with the same paramedic providing all chest compressions. The patient was a resuscitation manikin found in ventricular fibrillation (VF). The first shock terminated the VF and the patient remained in pulseless electrical activity (PEA) throughout the scenario. Average chest compression depth and rate was measured each minute for 12 minutes and divided into three groups based on chest compression quality; good (compression depth ≥ 40 mm, compression rate 100-120/minute for each minute of CPR), bad (initial compression depth 120/minute) or decay (change from good to bad during the 12 minutes). Changes in no-flow ratio (NFR, defined as the time without chest compressions divided by the total time of the ALS scenario) over time was also measured. Results Based on compression depth, 5 (26%), 9 (47%) and 5 (26%) were good, bad and with decay, respectively. Only one paramedic experienced decay within the first two minutes. Based on compression rate, 6 (32%), 6 (32%) and 7 (37%) were good, bad and with decay, respectively. NFR was 22% in both the 1-3 and 4-6 minute periods, respectively, but decreased to 14% in the 7-9 minute period (P = 0.002) and to 10% in the 10-12 minute period (P Conclusions In this simulated cardiac arrest manikin study, only half of the providers achieved guideline recommended compression depth during prolonged ALS. Large inter-individual differences in chest compression quality were already present from the initiation of CPR. Chest compression decay and thereby fatigue within the first two minutes was rare.
机译:背景技术这项研究的目的是测量心脏骤停模型中模拟的高级生命支持(ALS)期间的胸部按压衰减。方法19个护理人员小组,每人由3名护理人员组成,用同一名护理人员进行12分钟的ALS进行所有胸部按压。该患者是在室颤(VF)中发现的复苏假人。第一次电击终止了室颤,患者在整个情况下均保持无脉冲电活动(PEA)。每分钟测量一次平均胸部按压深度和速率,持续12分钟,并根据胸部按压质量将其分为三组;良好(压缩深度≥40毫米,CPR每分钟的压缩率100-120 /分钟),不良(初始压缩深度120 /分钟)或衰减(在12分钟内由好到坏变化)。还测量了无流量比率(NFR,定义为无胸部按压的时间除以ALS情景的总时间)随时间的变化。结果基于压缩深度,分别为好(5)(26%),坏(9)(47%)和衰变(5)(26%)。在最初的两分钟内,只有一名护理人员经历了衰变。基于压缩率,分别为6(32%),6(32%)和7(37%),好坏。在1-3分钟和4-6分钟内,NFR分别为22%,但在7-9分钟内下降到14%(P = 0.002),在10-12分钟内下降到10%(P结论在这项模拟的心脏骤停人体模型研究中,只有一半的提供者在长期ALS期间达到了指南推荐的按压深度;开始CPR时已经存在较大的个体间胸部按压质量差异;胸部按压衰减并因此导致前两次疲劳分钟是罕见的。

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