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Case report: an unstable wide QRS complexes tachycardia after ablation of a poster-septal accessory pathwayWhat is the mechanism?

机译:病例报告:后房间隔通路消融后不稳定的宽QRS波复合心动过速是什么机制?

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Introduction: Differentiation of wide QRS complex tachycardia required repeated electrophysiological stimuli and mapping. However, instability of tachycardia would increase the difficulty in differential diagnosis. Symptoms and Clinical findings: In this paper, we reported a wide QRS tachycardia following ablation of an atrioventricular reentrant tachycardia participated by a poster-septal accessory pathway . Limited differentiation strategy was performed because the wide QRS tachycardia was self-limited and with unstable hemodynamics. We analyzed the mechanism of the wide QRS tachycardia by only 4 beats ventricular overpacing. On the basis of the last ventricular pacing, an atypical atrioventricular nodal reentrant tachycardia was confirmed. Intervention and Outcomes: After slow-pathway modification, the wide QRS tachycardia was eliminated. Conclusion: It was an atypical atrial-ventricular node reentrant tachycardia with right bundle branch block. Reasonable analysis based on electrophysiological electrophysiologic knowledge was the basis of successful diagnosis and treatment.
机译:简介:宽QRS复杂性心动过速的分化需要反复的电生理刺激和映射。但是,心动过速的不稳定性会增加鉴别诊断的难度。症状和临床发现:在本文中,我们报道了由后房间隔辅助途径参与的房室折返性心动过速消融后出现了广泛的QRS心动过速。由于广泛的QRS心动过速是自限性的并且血液动力学不稳定,因此进行了有限的分化策略。我们仅通过4次心室过度搏动就分析了宽QRS心动过速的机制。在最后一次心室起搏的基础上,证实了非典型房室结折返性心动过速。干预和结果:慢路径修饰后,广泛的QRS心动过速被消除。结论:这是一种非典型的房室结折返性心动过速,右束支传导阻滞。基于电生理电生理知识的合理分析是成功诊断和治疗的基础。

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