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High Grade Atrioventricular Block Presenting with Cardiac Arrest

机译:高级别房室传导阻滞伴心脏骤停

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Introduction: Atrioventricular block usually does not cause cardiac arrest because of the development of an escape rhythm which maintains cardiac output. We report a case of high grade AV block presenting with cardiac arrest. Case Description: A 74-year-old man with past medical history of hypertension, dyslipidemia and a recent stroke was brought to the emergency room after a cardiac arrest, with pulseless electrical activity as the initial rhythm. Cardiopulmonary resuscitation was performed with return of spontaneous circulation after five minutes. On examination, he was unresponsive, with heart rate of 33 beats per minute, blood pressure of 108/51mmHg, normal heart sounds and clear lungs. He was given a total of 2mg of atropine, following which he was started on dopamine infusion, with no significant increase in heart rate. His electrocardiogram showed high grade AV block with ventricular rate of 30 beats per minute. An assessment of cardiac arrest due to severe conduction disease, with no evidence of acute coronary syndrome was made. A trans-venous pacemaker was inserted with improvement in the patient’s blood pressure and mental status. EKG revealed demand ventricular pacing with 100% ventricular capture. By the third day of admission, he was fully awake and following simple commands, but he remained pacemaker-dependent with no subsidiary rhythm. He had a dual chamber permanent pacemaker inserted without complication. Discussion: In advanced (high grade) second degree AV block, there is failure of conduction of two or more consecutive P waves. High grade AV block may be asymptomatic, or it may present with symptoms of hypoperfusion due to reduced cardiac output. Conclusion: This case describes a not previously reported presentation of high grade AV block with cardiac arrest and is in agreement with the 2008 American College of Cardiology/American Heart Association/Heart Rhythm Society (ACC/AHA/HRS) device guidelines recommendations for permanent pacemaker insertion.
机译:简介:房室传导阻滞通常不会导致心脏骤停,这是因为维持心律输出的逃逸节奏的发展。我们报告一例高级别房室传导阻滞伴心脏骤停。病例描述:一名有高血压,血脂异常和近期卒中病史的74岁男子在心脏骤停后被带到急诊室,最初的无搏动是电活动。五分钟后进行心肺复苏并恢复自发性循环。经检查,他无反应,心律为每分钟33次,血压为108 / 51mmHg,心音正常,肺部通畅。给予他总共2mg的阿托品,然后开始多巴胺输注,但心率没有明显增加。他的心电图显示高级别的房室传导阻滞,心室速率为每分钟30次。没有严重急性冠脉综合征的证据,评估了由严重传导疾病引起的心脏骤停。插入经静脉起搏器可改善患者的血压和精神状态。心电图显示需求心律起搏,心室捕获率为100%。入院的第三天,他已经完全清醒并遵循简单的命令,但是他仍然依赖于起搏器,没有副节奏。他插入了一个双腔永久性起搏器,无任何并发症。讨论:在高级(高级)二级房室传导阻滞中,两个或多个连续的P波传导失败。高级别的房室传导阻滞可能是无症状的,或者由于心排血量减少而出现灌注不足的症状。结论:该病例描述了先前未报道的伴有心脏骤停的高级房室传导阻滞,并与2008年美国心脏病学会/美国心脏协会/心律协会(ACC / AHA / HRS)关于永久性起搏器的设备指南建议一致插入。

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