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首页> 外文期刊>Advances in Interventional Cardiology: Postepy w Kardiologii Interwencyjnej >Cardiac rupture in takotsubo cardiomyopathy treated surgically
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Cardiac rupture in takotsubo cardiomyopathy treated surgically

机译:takotsubo心肌病的心脏破裂手术治疗

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A 74-year-old woman, without a history of cardiological problems or risk factors of cardiovascular diseases, was admitted to the emergency room of our hospital after 2 h of chest pain. During the transport, the patient received 5000 U of unfractionated heparin, aspirin (300 mg) and clopidogrel (600 mg). The patient had suffered from an anxiety syndrome for several years. At admission, the patient was in a serious condition generally, was vomiting, and had severe chest pain with signs of cardiogenic shock (skin pale and wet, blood presure (BP) and heart rate (HR) undetectable). Electrocardiogram demonstrated a sinus rhythm of 58 per minute with QS complex and ST segment elevation in precordial leads (V2–V6). Laboratory results revealed increased troponin I concentration (2.041 ng/ml). Following hemodynamic stabilization, the patient was transported to the catheterization laboratory. Coronarography did not reveal any significant stenosis. Left ventricle angiography (LVA) showed normal volume with contractile disturbances of apex and hyperkinesis of the basement segments, with ejection fraction (EF) of 56%. Contrast outflow to the epicardium was observed within the area of the apex, through the perforated wall of the left ventricle (Figures 1 A–C). Echo confirmed the presence of fluid in the pericardium and cardiac tamponade. The patient was supported with intra-aortic balloon contra-pulsation and transported to the cardiac surgery for urgent intervention. During transport, the patient lost consciousness. After urgent cardiac tamponade decompression, the pulse and arterial pressure increased. Active bleeding through the ruptured left ventricle was observed in the area of the apex during the operation. Left ventricular plication with sutures on a double layered Teflon pad was performed. The lines of the sutures were conducted through healthy tissues. Examination of the supported ruptured region showed left ventricle tightness and complete hemostasis. No significant complications were observed during the perioperative period. On the first day after the operation the patient was extubated, and on the fifth day the intra-aortic balloon contra-pulsation was removed. The patient was transported to the regional hospital on day 11 to continue the therapeutic and rehabilitation procedures. A discharge echocardiogram revealed akinesis of the apex and hypokinesis of the septum, with an EF of 50%. The patient was under cardiosurgical follow-up for the next 3... View full text...
机译:一名74岁的女性,无心脏病史或心血管疾病危险因素,在胸痛2小时后被送入我院急诊室。在运输过程中,患者接受了5000 U普通肝素,阿司匹林(300毫克)和氯吡格雷(600毫克)。该患者患有焦虑症已有数年。入院时,患者一般情况较重,正在呕吐,并伴有严重的胸痛,并伴有心源性休克的迹象(皮肤苍白潮湿,无法检测到血液压力(BP)和心率(HR))。心电图显示窦性心律为每分钟58分钟,心电图导联(V2-V6)的QS复杂度和ST段抬高。实验室结果显示肌钙蛋白I浓度增加(2.041 ng / ml)。血液动力学稳定后,将患者转移到导管实验室。冠状动脉造影未发现任何明显的狭窄。左心室血管造影(LVA)表现为正常容量,具有收缩收缩性先兆和基底节运动亢进,射血分数(EF)为56%。在心尖区域,通过左心室的穿孔壁观察到相反的流向心外膜(图1 A–C)。回声证实心包和心脏压塞中存在液体。该患者受到主动脉内球囊反搏的支持,并被送至心脏外科进行紧急干预。在运输过程中,患者失去知觉。心脏压塞紧急减压后,脉搏和动脉压升高。在手术过程中,在顶点区域观察到通过破裂的左心室活动性出血。在双层聚四氟乙烯垫上进行缝合的左心室褶皱。缝合线通过健康组织进行。检查支持的破裂区域显示左心室紧闭和完全止血。围手术期未见明显并发症。术后第一天拔管,第五天去除主动脉内球囊反搏。患者在第11天被运送到地区医院以继续治疗和康复程序。超声心动图检查显示房顶运动不全和隔膜运动减退,EF为50%。该患者在接下来的3个月接受了心脏外科手术的随访。查看全文...

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