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首页> 外文期刊>Acta Stomatologica Croatica >Mandibular Osteonecrosis due to the Pulpal-Periodontal Syndrome: a Case Report and Review of the Literature
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Mandibular Osteonecrosis due to the Pulpal-Periodontal Syndrome: a Case Report and Review of the Literature

机译:下颌骨牙周综合征引起的下颌骨坏死:一例报道并文献复习

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Objective: Ishemic bone disease has multifactorial etiologies. Cronic dental infections should be eliminated to prevent osteonecrosis of the jaw. Case report: We report an unusual case of osteonecrosis due to the pulpal-peridontal syndrome and subsequent pulp necrosis. A case of 38 year old woman who presented with exposed bone, 8 mm in diameter, in the lingual area of the right lower third molar. The patient was otherwise healthy and was not taking any medications. A detailed medical history showed no previous diseases. Patient denied any type of local trauma. A complete blood count showed no abnormalities. The panoramic radiograph revealed a deep periodontal pocket between teeth 47 and 48. The CBCT revealed a deep periodontal pocket between molars and bone sequestrum of the lingual plate. Topical treatment consisted of adhesive periodontal dressing based on the cellulose and betamethasone oitnment together with orabase, without improvement. Therefore, peroral amoxycillin was prescribed for a week. Since there was no improvement, the third molar was removed as well as necrotic bone; the alveolar bone was remodelled and utures were placed. After suturing, the whole area was covered using intraoral resorbable bandage. Microbial swab of the wound aspirate did not reveal polymorphonuclears or the presence of icroorganisms. Microbial swab of the biopsy specimen of the necrotic bone particle and sequestrum showed a large amount of gram-positive coccae, however, polymorphonuclears were not found. Histopathological analysis revealed acute chronic inflammation. One week after the surgery, the area healed completely. Conclusion: This case highlights the fact that in some patients bone exposure might develop due to the pulpal-peridontal syndrome i.e. pulp necrosis.
机译:目的:缺血性骨病具有多种病因。应该消除早期的牙齿感染,以防止颌骨坏死。病例报告:我们报告了因牙周周综合症和随后的牙髓坏死引起的骨坏死异常病例。一例38岁女性,右下颌第三磨牙的舌头区域露出直径8毫米的裸露骨。否则患者健康,没有服用任何药物。详细的病史没有发现先前的疾病。病人否认有任何形式的局部创伤。全血细胞计数未发现异常。全景放射线照片显示在牙齿47和48之间有一个深的牙周袋。CBCT显示了在磨牙和舌板的死骨之间有一个深的牙周袋。局部治疗由基于纤维素和倍他米松软膏以及orabase的牙周贴敷而没有改善。因此,口服阿莫西林的处方为一周。由于没有改善,去除了第三磨牙以及坏死骨。牙槽骨重塑并放置缝合线。缝合后,使用口内可吸收绷带覆盖整个区域。伤口抽吸物的微生物拭子未发现多形核或微生物的存在。坏死骨颗粒和死骨的活检标本的微生物拭子显示出大量的革兰氏阳性球菌,但未发现多形核。组织病理学分析显示急性慢性炎症。手术一周后,该区域完全愈合。结论:该病例突出了以下事实:在某些患者中,由于牙髓-牙周综合症,即牙髓坏死,可能会导致骨骼暴露。

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