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Non-traumatic fractures following seizures: two case reports

机译:癫痫发作后的非创伤性骨折:两例病例报告

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Introduction Seizures with or without trauma may cause fractures that occur commonly in epileptic seizures. Fracture risk is less reported in non-epileptic seizures. Some metabolic conditions leading to a decrease in bone mineral density may cause fractures secondary to non-epileptic seizure. Case presentation We describe two cases of non-traumatic acetabular and vertebrae fractures following seizures without history of epilepsy. They occurred in two male patients, 18 and 48 years old suffering respectively from hypercorticism and poorly controlled diabetes mellitus. Seizures, occurring inside hospital, were secondary to hypertensive encephalopathy crisis with hypokaliemia in the first case and severe hypoglycaemia in the second one. Fracture was promoted by a decrease in mineral bone density caused respectively by hypercorticism and diabetic chronic renal failure. Conclusion These observations emphasize that fracture prevention among patients with decreased mineral bone density should include the avoidance of metabolic causes of seizure.
机译:简介有或无外伤的癫痫发作都可能导致骨折,通常发生在癫痫性癫痫发作中。非癫痫发作的骨折风险较少。导致骨矿物质密度降低的某些代谢条件可能会导致继发于非癫痫性发作的骨折。病例介绍我们描述了两例非创伤性髋臼和椎骨骨折,发作后没有癫痫病史。它们发生在两名分别患有皮质激素过多和糖尿病控制不良的18岁和48岁男性患者中。发生在医院内的癫痫发作是继发于高血压脑病危机的,第一例为低钾血症,第二例为严重低血糖。皮质过度增高和糖尿病慢性肾功能衰竭引起的矿物质骨密度降低促进骨折。结论这些观察结果强调,在矿物质骨密度降低的患者中预防骨折应包括避免代谢性发作的原因。

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