Small Molecule TH-39 Potentially Targets Hec1/Nek2 Interaction and Exhibits Antitumor Efficacy in K562 Cells via G0/G1 Cell Cycle Arrest and Apoptosis Induction

Yongxia Zhu; Wei Wei; Tinghong Ye; Zhihao Liu; Li Liu; Yong Luo; Lidan Zhang; Chao Gao; Ningyu Wang; Luoting Yu

首页> 外文期刊>Cellular Physiology and Biochemistry >Small Molecule TH-39 Potentially Targets Hec1/Nek2 Interaction and Exhibits Antitumor Efficacy in K562 Cells via G0/G1 Cell Cycle Arrest and Apoptosis Induction

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Small Molecule TH-39 Potentially Targets Hec1/Nek2 Interaction and Exhibits Antitumor Efficacy in K562 Cells via G0/G1 Cell Cycle Arrest and Apoptosis Induction

机译：小分子TH-39潜在地靶向Hec1 / Nek2相互作用，并通过G0 / G1细胞周期阻滞和凋亡诱导作用在K562细胞中表现出抗肿瘤功效

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《Cellular Physiology and Biochemistry》 |2016年第2期|共12页
作者
Yongxia Zhu; Wei Wei; Tinghong Ye; Zhihao Liu; Li Liu; Yong Luo; Lidan Zhang; Chao Gao; Ningyu Wang; Luoting Yu;
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中图分类细胞生物物理学;
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7. Small Molecule TH-39 Potentially Targets Hec1/Nek2 Interaction and Exhibits Antitumor Efficacy in K562 Cells via G0/G1 Cell Cycle Arrest and Apoptosis Induction [O] . Yongxia Zhu, Wei Wei, Tinghong Ye, 2016

机译：小分子TH-39潜在地靶向Hec1 / Nek2相互作用并通过G0 / G1细胞周期阻滞和凋亡诱导在K562细胞中表现出抗肿瘤功效

Small Molecule TH-39 Potentially Targets Hec1/Nek2 Interaction and Exhibits Antitumor Efficacy in K562 Cells via G0/G1 Cell Cycle Arrest and Apoptosis Induction

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