Essential role of microRNA-155 in the pathogenesis of autoimmune arthritis in mice

Stephan Blüml; Michael Bonelli; Birgit Niederreiter; Antonia Puchner; Georg Mayr; Silvia Hayer; Marije I. Koenders; Wim B. van den Berg; Josef Smolen; Kurt Redlich

首页> 外文期刊>Arthritis & Rheumatism >Essential role of microRNA-155 in the pathogenesis of autoimmune arthritis in mice

【24h】

Essential role of microRNA-155 in the pathogenesis of autoimmune arthritis in mice

机译：microRNA-155在小鼠自身免疫性关节炎发病机理中的重要作用

获取原文

获取原文并翻译 | 示例

获取外文期刊封面封底 >>

开具论文收录证明 >>

文献代查 >>

页面导航

摘要
著录项
相似文献
相关主题

摘要

MicroRNAs (miRNA) are a new class of regulatory elements. Altered expression of miRNA has been demonstrated in the inflamed joints of patients with rheumatoid arthritis (RA). The aim of this study was to examine the role of miRNA in the pathogenesis of autoimmune arthritis, using 2 murine models.

机译：微小RNA（miRNA）是一类新的调控元件。在类风湿性关节炎（RA）患者的发炎关节中已证明miRNA的表达发生了改变。这项研究的目的是使用2种小鼠模型研究miRNA在自身免疫性关节炎发病机理中的作用。

著录项

来源
《Arthritis & Rheumatism》 |2011年第5期|1281-1288|共8页
作者
Stephan Blüml; Michael Bonelli; Birgit Niederreiter; Antonia Puchner; Georg Mayr; Silvia Hayer; Marije I. Koenders; Wim B. van den Berg; Josef Smolen; Kurt Redlich;
展开▼
作者单位

Medical University of Vienna Vienna Austria;

Radboud University Nijmegen Medical Center Nijmegen The Netherlands;

展开▼
收录信息
原文格式 PDF
正文语种 eng
中图分类
关键词

相似文献

外文文献
中文文献
专利

1. Essential role of microRNA-155 in the pathogenesis of autoimmune arthritis in mice. [J] . Bluml S, Bonelli M, Niederreiter B, Arthritis and Rheumatism . 2011,第5期

机译：microRNA-155在小鼠自身免疫性关节炎发病机理中的重要作用。
2. The role of nitric oxide in the pathogenesis of spontaneous murine autoimmune disease: increased nitric oxide production and nitric oxide synthase expression in MRL-lpr/lpr mice, and reduction of spontaneous glomerulonephritis and arthritis by orally administered NG-monomethyl-L-arginine. [J] . J B Weinberg, D L Granger, D S Pisetsky, The Journal of Experomental Medicine . 1994,第2期

机译：一氧化氮在自发鼠源性自身免疫疾病发病机理中的作用：通过口服NG-单甲基-L-精氨酸，增加MRL-lpr / lpr小鼠中一氧化氮的产生和一氧化氮合酶的表达，并减少自发性肾小球肾炎和关节炎。
3. Role of Th17 and Treg cells in septic arthritis and the impact of the Th17/Treg -derived cytokines in the pathogenesis of ce:italic>S. aureus/ce:italic> induced septic arthritis in mice [J] . Ipsita Dey, Biswadev Bishayi Microbial Pathogenesis . 2017,第期

机译：Th17和Treg细胞在脓肠梗性关节炎中的作用及Th17 / Treg-Derived细胞因子在＆ Ce：斜体的发病机制中的影响。金黄色葡萄球菌＆ / ce：斜体>诱导小鼠的化粪池关节炎
4. A novel model for pathogenesis of autoimmune heart failure: The role of dendritic cells [C] . Manu Rangachari, Urs Eriksson, Elisabeth H. Vollmann, Uehara Memorial Foundation Symposium on the Innate Immune System . 2005

机译：自身免疫性心力衰竭发病机制的新模型：树突细胞的作用
5. Exploring the Role of Gut Microbiota as Modifiers of Autoimmune Pathogenesis in Interleukin-10 Deficient Mice [D] . Bobe, Alexandria Marie. 2017

机译：旨在肠道微生物达作为白细胞介素-10缺陷小鼠自身免疫发病机制的改性的作用
6. The role of nitric oxide in the pathogenesis of spontaneous murine autoimmune disease: increased nitric oxide production and nitric oxide synthase expression in MRL-lpr/lpr mice and reduction of spontaneous glomerulonephritis and arthritis by orally administered NG-monomethyl-L- arginine [O] . 1994

机译：一氧化氮在自发鼠源性自身免疫性疾病发病机理中的作用：MRL-lpr / lpr小鼠中一氧化氮的产生和一氧化氮合酶的表达增加以及口服NG-单甲基-L-精氨酸可减少自发性肾小球肾炎和关节炎
7. The role of nitric oxide in the pathogenesis of spontaneous murine autoimmune disease: increased nitric oxide production and nitric oxide synthase expression in MRL-lpr/lpr mice, and reduction of spontaneous glomerulonephritis and arthritis by orally administered NG-monomethyl-L- arginine [O] . 1994

机译：一氧化氮在自发鼠源性自身免疫性疾病发病机理中的作用：MRL-lpr / lpr小鼠中一氧化氮的产生和一氧化氮合酶的表达增加，以及口服NG-单甲基-L-精氨酸可减少自发性肾小球肾炎和关节炎

Essential role of microRNA-155 in the pathogenesis of autoimmune arthritis in mice

摘要

著录项

相似文献

相关主题

期刊订阅