机译:X框绑定蛋白1和人类囊性纤维化肺泡巨噬细胞的天然免疫反应。
Marsico Lung Institute/Cystic Fibrosis Research Center, The University of North Carolina at Chapel Hill, 125 Mason Farm Road, CB #7248, Chapel Hill, NC 27599-7248;
Marsico Lung Institute/Cystic Fibrosis Research Center, The University of North Carolina at Chapel Hill, Chapel Hill, North Carolina;
Marsico Lung Institute/Cystic Fibrosis Research Center, The University of North Carolina at Chapel Hill, Chapel Hill, North Carolina,Department of Medicine, The University of North Carolina at Chapel Hill, Chapel Hill, North Carolina;
Marsico Lung Institute/Cystic Fibrosis Research Center, The University of North Carolina at Chapel Hill, Chapel Hill, North Carolina,Department of Medicine, The University of North Carolina at Chapel Hill, Chapel Hill, North Carolina;
Marsico Lung Institute/Cystic Fibrosis Research Center, Department of Medicine, Department of Cell Biology and Physiology, The University of North Carolina at Chapel Hill, 125 Mason Farm Road, CB #7248, Chapel Hill, NC 27599-7248;
cystic fibrosis; airway inflammation; alveolar macrophage; UPR; IRE1α/XBP-1;
机译:炭疽芽孢杆菌孢子通过多种促分裂原激活的蛋白激酶途径刺激人肺泡巨噬细胞中的细胞因子和趋化因子先天性免疫反应。
机译:囊性纤维化气道细胞中未折叠蛋白反应的非典型激活有助于p38 MAPK介导的先天免疫反应
机译:囊性纤维化气道细胞中未折叠的蛋白应答的非典型激活有助于p38 MAPK介导的先天免疫应答。
机译:肺炎肺巨噬细胞先天免疫反应的最佳控制
机译:先天性免疫收集蛋白表面活性蛋白D和适应性免疫蛋白IgM对增强肺泡巨噬细胞清除晚期凋亡细胞的作用
机译:X盒结合蛋白1和人类囊性纤维化肺泡巨噬细胞的天然免疫反应。
机译:炭疽芽孢杆菌孢子通过多种促分裂原活化的蛋白激酶途径刺激人肺泡巨噬细胞的细胞因子和趋化因子先天免疫反应†
机译:炭疽致死毒素损害肺泡巨噬细胞的先天免疫功能,促进炭疽杆菌存活