机译:缺损内皮细胞之间的开口说明肿瘤血管渗漏
From the Cardiovascular Research Institute and Department of Anatomy,University of California, San Francisco, California;
From the Cardiovascular Research Institute and Department of Anatomy,University of California, San Francisco, California;
From the Cardiovascular Research Institute and Department of Anatomy,University of California, San Francisco, California;
From the Cardiovascular Research Institute and Department of Anatomy,University of California, San Francisco, California;
From the Cardiovascular Research Institute and Department of Anatomy,University of California, San Francisco, California;
and the Edwin L. Steele Laboratory,Department of Radiation Oncology, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusetts;
and the Edwin L. Steele Laboratory,Department of Radiation Oncology, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusetts;
From the Cardiovascular Research Institute and Department of Anatomy,University of California, San Francisco, California;
机译:表达Bcl-2的内皮细胞通过增强肿瘤血管生成,血管渗漏和肿瘤侵袭来促进肿瘤转移
机译:肿瘤中血管内皮生长因子(VEGF)表达的条件转换:VEGF撤药诱导内皮细胞脱落和血管母细胞瘤样血管消退
机译:靶向肿瘤相关的内皮细胞:抗VEGFR2免疫脂质体介导肿瘤血管破裂并抑制肿瘤生长。
机译:鸡绒膜尿囊膜共聚焦观察血管间质和肿瘤细胞内膜内皮细胞中亲水性和亲脂性光敏剂
机译:肿瘤细胞与血管系统相互作用的分子研究:粘附分子,内皮细胞收缩,肿瘤细胞扩散和类花生酸12(S)-HETE的调节作用。
机译:缺损内皮细胞之间的开口说明肿瘤血管渗漏
机译:缺损内皮细胞之间的开口说明肿瘤血管渗漏