机译:缺乏12/15脂氧合酶可减少载脂蛋白E缺乏症小鼠的脑氧化应激
From the Department of Pharmacology,University of Pennsylvania School of Medicine, Philadelphia, Pennsylvania;
From the Department of Pharmacology,University of Pennsylvania School of Medicine, Philadelphia, Pennsylvania;
From the Department of Pharmacology,University of Pennsylvania School of Medicine, Philadelphia, Pennsylvania;
and the Departments of Physiology and Biochemistry,Queen’s University, Kingston, Ontario, Canada;
From the Department of Pharmacology,University of Pennsylvania School of Medicine, Philadelphia, Pennsylvania;
机译:缺乏12 / 15-脂加氧酶表达可减少载脂蛋白e缺乏症小鼠的脂质过氧化和动脉粥样硬化。
机译:载脂蛋白E缺乏症小鼠中血栓烷受体拮抗作用与12/15脂氧合酶基因破坏的相加抗动脉粥样硬化作用。
机译:12 / 15-脂氧合酶基因的破坏和维生素E的施用通过最终的通用途径减少了载脂蛋白E缺乏症小鼠的动脉粥样硬化和氧化应激
机译:来自Polygonum Cuspidatum的抗氧化剂白藜芦醇在SAMP8小鼠中逆转记忆障碍和脑氧化应激
机译:人血小板12-脂氧合酶,人网织细胞15-脂氧合酶-1和人上皮15-脂氧合酶-2的变构,机理和抑制研究。
机译:缺乏12/15脂氧合酶可减少载脂蛋白E缺乏症小鼠的脑氧化应激
机译:缺乏12/15脂氧合酶可减少载脂蛋白E缺乏症小鼠的脑氧化应激