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Disinhibited Attachment Disorder in UK Adopted Children During Middle Childhood: Prevalence Validity and Possible Developmental Origin

机译:英国收养儿童中期成瘾的解除依恋障碍:患病率有效性和可能的​​发育起源

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摘要

We investigate the prevalence, specificity and possible aetiology of Disinhibited Attachment Disorder (DAD) in adopted children without a history of institutional care. Sixty children adopted from UK out-of-home care (AD; mean age 102 months, 45 % male); 26 clinic-referred children with externalizing disorder (ED; mean age 104 months, 77 % male) but no history of maltreatment or disrupted care; and 55 matched low-risk comparison controls (LR; mean age 108 months, 49 % male) were assessed for DAD using a triangulation of parent, teacher, and research observations. Maltreatment history and child psychiatric symptoms were obtained from parent report and child language development was assessed. DAD was identified in 49 % of AD, 4 % of ED and 6 % of LR children. Seventy-two percent of AD children had suffered maltreatment. DAD was not associated with degree of risk exposure, demographics, or language. A significant association with ADHD did not explain variance in DAD prevalence across groups. DAD was significantly more common in children first admitted to out-of-home care between 7 and 24 months, independent of maltreatment severity, age at adoption and number of care placements. Implications for developmental theory, adoption policy and clinical application are discussed.Electronic supplementary materialThe online version of this article (doi:10.1007/s10802-016-0131-2) contains supplementary material, which is available to authorized users.
机译:我们调查在没有机构护理史的收养儿童中,禁忌型依恋障碍(DAD)的患病率,特异性和可能的​​病因。 60名从英国家庭外照看的儿童(AD;平均年龄102个月,男性占45%); 26名临床转诊的患有外在性疾病的儿童(ED;平均年龄104个月,男性77%),但没有虐待或护理中断的病史;使用父母,老师和研究观察的三角剖分法,评估了55个匹配的低风险比较对照(LR;平均年龄108个月,男性49%)的DAD。从父母的报告中获得虐待历史和儿童精神症状,并评估儿童的语言发展。在49%的AD,4%的ED和6%的LR儿童中发现DAD。 72%的AD儿童遭受过虐待。 DAD与风险暴露程度,人口统计学或语言无关。与ADHD的显着相关性不能解释各组DAD患病率的差异。 DAD在7到24个月之间首次入院的儿童中更为普遍,不受虐待严重程度,收养年龄和护理地点数量的影响。电子补充材料本文的在线版本(doi:10.1007 / s10802-016-0131-2)包含补充材料,授权用户可以使用。

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