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Hypoxia-Induced Phenotypic Transformation of Corpus Cavernosum Smooth Muscle Cells After Cavernous Nerve Crush Injury by Down-Regulating P38 Mitogen-Activated Protein Kinase Expression

机译：下调P38丝裂原活化蛋白激酶表达对缺氧诱导海绵体神经挤压损伤后海绵体平滑肌细胞表型的转化

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摘要

Cavernosal nerve (CN) injury is commonly caused by radical prostatectomy surgery, and it might directly lead to erectile dysfunction (ED). Currently, the role of mitogen-activated protein kinase (MAPK) family proteins in phenotypic transformation of corpus cavernosum smooth muscle cell (CCSMC) after CNs injury is poorly understood.

机译：海绵体神经（CN）损伤通常由前列腺癌根治术引起，可能直接导致勃起功能障碍（ED）。目前，对CNs损伤后丝裂原活化蛋白激酶（MAPK）家族蛋白在海绵体平滑肌细胞（CCSMC）表型转化中的作用了解甚少。

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期刊名称 Sexual Medicine
作者
Sixiang Chen; Xiaojun Huang; Xianghui Kong; Zhaohui Sun; Fan Zhao; Wenjie Huang; Miaoyong Ye; Ke Ma; Tingting Tao; Bodong Lv;
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作者单位

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年(卷),期 2019(7),4
年度 2019
页码 -1
总页数 8
原文格式 PDF
正文语种
中图分类生理学 ;
关键词
Erectile Dysfunction; Neurogenic; Phenotypic Transformation; Mitogen-Activated Protein Kinase (MAPK); p38 MAPK; Hypoxia;

机译：勃起功能障碍;神经源性;表型转化;丝裂原激活的蛋白激酶（MAPK）;p38 MAPK;缺氧;

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专利

1. The Protective Effect of Salidroside on Hypoxia-Induced Corpus Cavernosum Smooth Muscle Cell Phenotypic Transformation [J] . Zhang Xiang, Zhao Jian-feng, Zhao Fan, Evidence-based complementary and alternative medicine: eCAM . 2017 ,第7期

机译：Salidroside对缺氧诱导的患有肺肿瘤平滑肌细胞表型转化的保护作用
2. Maintenance of the contractile phenotype in corpus cavernosum smooth muscle cells by Myocardin gene therapy ameliorates erectile dysfunction in bilateral cavernous nerve injury rats [J] . Zhang H. -B., Wang Z. -Q., Chen F. -Z., Andrology . 2017 ,第4期

机译：Myocardin基因治疗的Corpus Cavernosum平滑肌细胞中的收缩表型的维护改善了双侧海绵神经损伤大鼠的勃起功能障碍
3. Transforming growth factor-β-induced alpha-smooth muscle cell actin expression in renal proximal tubular cells is regulated by p38β mitogen-activated protein kinase, extracellular signal-regulated protein kinase1,2 and the Smad signalling during epithelial–myofibroblast transdifferentiation [J] . Attila Sebe12 Suvi-Katri Leivonen35 Attila Fintha12 Andras Masszi12 Laszlo Rosivall12 Veli-Matti Kähäri3 and Istvan Mucsi24 Nephrology Dialysis Transplantation . 2008 ,第5期

机译：上皮细胞-成纤维细胞转分化过程中，转化生长因子-β诱导的肾近端肾小管细胞中的α-平滑肌细胞肌动蛋白表达受到p38β丝裂原活化蛋白激酶，细胞外信号调节蛋白激酶1,2和Smad信号传导的调节
4. Activation of Mitogen-activated Protein Kinases and Protein Kinase B/akt Signaling By Oxidative Stress in Vascular Smooth Muscle Cells: involvement in Vascular Pathophysiology [C] . Ashok K. Srivastava, Nihar R. Pandey, Antoine Blanc International Society for Heart Research.Congress . 2004

机译：血管平滑肌细胞氧化应激激活丝裂剂活化蛋白激酶和蛋白激酶B / Akt信号传导：血管病理生理学中的参与
5. Chronic ethanol exposure alters expression of insulin-induced mitogen-activated protein kinases in hypertensive vascular smooth muscle cells. [D] . Williams, Sparkle D. 2010

机译：慢性乙醇暴露会改变高血压血管平滑肌细胞中胰岛素诱导的促分裂原活化蛋白激酶的表达。
6. The Protective Effect of Salidroside on Hypoxia-Induced Corpus Cavernosum Smooth Muscle Cell Phenotypic Transformation [O] . Xiang Zhang, Jian-feng Zhao, Fan Zhao, 2017

机译：红景天苷对缺氧诱导的海绵体平滑肌细胞表型转化的保护作用
7. Hypoxia-Induced Phenotypic Transformation of Corpus Cavernosum Smooth Muscle Cells After Cavernous Nerve Crush Injury by Down-Regulating P38 Mitogen-Activated Protein Kinase Expression [O] . Sixiang Chen, Xiaojun Huang, Xianghui Kong, 2019

机译：通过下调节P38丝裂型活化蛋白激酶表达，缺氧诱导的心脏挤压损伤后囊腔探伤肌细胞的表型转化

Hypoxia-Induced Phenotypic Transformation of Corpus Cavernosum Smooth Muscle Cells After Cavernous Nerve Crush Injury by Down-Regulating P38 Mitogen-Activated Protein Kinase Expression

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