首页> 美国卫生研究院文献>OncoTargets and therapy >miR-1269b Drives Cisplatin Resistance of Human Non-Small Cell Lung Cancer via Modulating the PTEN/PI3K/AKT Signaling Pathway

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miR-1269b Drives Cisplatin Resistance of Human Non-Small Cell Lung Cancer via Modulating the PTEN/PI3K/AKT Signaling Pathway

机译：miR-1269b通过调节PTEN / PI3K / AKT信号通路驱动人非小细胞肺癌的顺铂耐药性

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摘要

MiRNAs have been reported to induce certain drug resistance in multiple solid tumors via various mechanisms. Our study aimed to investigate whether miRNA-1269b was involved in the chemoresistance and the progression of non-small cell lung cancer (NSCLC).

机译：据报道，miRNA通过多种机制在多种实体瘤中诱导一定的耐药性。我们的研究旨在调查miRNA-1269b是否参与化学耐药性和非小细胞肺癌（NSCLC）的进展。

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期刊名称 OncoTargets and therapy
作者
Wu Yang; Wei Xiao; Zhengrong Cai; Shidai Jin; Tian Li;
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作者单位

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年(卷),期 2020(13),-1
年度 2020
页码 -1
总页数 10
原文格式 PDF
正文语种
中图分类应用微生物学;肿瘤学;
关键词
miR-1269b; NSCLC; cisplatin resistant; PTEN; PI3K/AKT signaling;

机译：miR-1269b;NSCLC;顺铂耐药;PTEN;PI3K / AKT信号传导;

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专利

1. miR-1269b Drives Cisplatin Resistance of Human Non-Small Cell Lung Cancer via Modulating the PTEN/PI3K/AKT Signaling Pathway [J] . Wu Yang, Wei Xiao, Zhengrong Cai, OncoTargets and therapy . 2020,第2期

机译：MIR-1269B通过调节PTEN / PI3K / AKT信号通路驱动人非小细胞肺癌的顺铂抗性
2. Long non-coding RNA CASC2 enhanced cisplatin-induced viability inhibition of non-small cell lung cancer cells by regulating the PTEN/PI3K/Akt pathway through down-regulation of miR-18a and miR-21 [J] . Li Li, Zhang Haifeng, Wang Xiaolong, RSC Advances . 2018,第29期

机译：通过调节MIR-18A和MIR-21的下调，通过调节PTEN / PI3K / AKT途径来增强连铂诱导的非小细胞肺癌细胞的活力抑制非小细胞肺癌细胞
3. Kinesin family member 2A promotes cancer cell viability, mobility, stemness, and chemoresistance to cisplatin by activating the PI3K/AKT/VEGF signaling pathway in non-small cell lung cancer [J] . Liwei Xu, Xiaochun Zhang, Zanfeng Wang, American Journal of Translational Research . 2021,第4期

机译：通过在非小细胞肺癌中激活PI3K / AKT / VEGF信号传导途径来促进Cisplatin的癌细胞活力，迁移率，茎和化学抑制剂，促进癌细胞活力，迁移率，茎和化学抑制性
4. THE E3 UBIQUITIN LIGASE CBL-B REVERSES MULTIDRUG RESISTANCE OF MCF-7/ADR CELLS THROUGH SUPPRESSION OF PI3K/AKT SIGNALING PATHWAY AND DOWN-REGULATION OF P-GLYCOPROTEIN [C] . Ye Zhang, Xiujuan Qu, Xuejun Hu, 2011 Asia-Pacific Conference of tumor biology and medicine . 2011

机译：E3泛素连接酶CBL-B通过抑制PI3K / AKT信号通路和P-糖蛋白下调逆转MCF-7 / ADR细胞的多药耐药性
5. Sensitivity of MCF-7 breast cancer cells to anticancer drugs is decreased by activation of the PI3K/PDK/Akt signal transduction pathway. [D] . Navolanic, Patrick M. 2004

机译：通过激活PI3K / PDK / Akt信号转导途径，MCF-7乳腺癌细胞对抗癌药物的敏感性降低。
6. miR-181 regulates cisplatin-resistant non-small cell lung cancer via downregulation of autophagy through the PTEN/PI3K/AKT pathway [O] . Jiangang Liu, Yingru Xing, Ling Rong -1

机译：miR-181通过下调PTEN / PI3K / AKT途径的自噬来调节顺铂耐药的非小细胞肺癌
7. >miR-1269b Drives Cisplatin Resistance of Human Non-Small Cell Lung Cancer via Modulating the PTEN/PI3K/AKT Signaling Pathway [O] . Wu Yang, Wei Xiao, Zhengrong Cai, 2020

机译：> MiR-1269B通过调节PTEN / PI3K / AKT信号通路（PTEN / P）驱动人非小细胞肺癌的顺铂抗性

miR-1269b Drives Cisplatin Resistance of Human Non-Small Cell Lung Cancer via Modulating the PTEN/PI3K/AKT Signaling Pathway

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