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Identification of age-specific Nrf2 binding to a novel ARE locus in the Gclc promoter: a compensatory means for the loss of glutathione synthetic capacity in the aging rat liver?

机译：鉴定与新型新型的年龄特异性NRF2结合是GCLC启动子的基因座：用于衰老大鼠肝脏谷胱甘肽合成能力的补偿手段？

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期刊名称 other
作者
Swapna V. Shenvi; Eric Smith; Tory M. Hagen;
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作者单位

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年(卷),期 -1(11),2
年度 -1
页码 297–304
总页数 20
原文格式 PDF
正文语种
中图分类
关键词
Antioxidant Response Element (ARE) Bach1 small maf proteins CREB-binding protein (CBP) Glutamate cysteine ligase catalytic subunit (GCLC);

机译：抗氧化反应元件（ARE）;Bach1;小maf蛋白;CREB结合蛋白（CBP）;谷氨酸半胱氨酸连接酶催化亚基（GCLC）;

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1. Identification of age-specific Nrf2 binding to a novel antioxidant response element locus in the Gclc promoter: a compensatory means for the loss of glutathione synthetic capacity in the aging rat liver? [J] . Swapna V Shenvi, Eric Smith, Tory M Hagen Aging cell. . 2012,第2期

机译：鉴定年龄特异性Nrf2与Gclc启动子中新型抗氧化反应元件基因座的结合：衰老大鼠肝脏中谷胱甘肽合成能力丧失的一种补偿手段？
2. Identification of age-specific Nrf2 binding to a novel antioxidant response element locus in the Gclc promoter: a compensatory means for the loss of glutathione synthetic capacity in the aging rat liver? [J] . Swapna V. Shenvi, Eric Smith, Tory M. Hagen Aging Cell . 2012,第2期

机译：鉴定年龄特异性Nrf2与Gclc启动子中新型抗氧化反应元件基因座的结合：衰老大鼠肝脏中谷胱甘肽合成能力丧失的一种补偿手段？
3. Identification of age-specific Nrf2 binding to a novel antioxidant response element locus in the Gclc promoter: a compensatory means for the loss of glutathione synthetic capacity in the aging rat liver? [J] . Swapna V Shenvi, Eric Smith, Tory M Hagen Aging cell. . 2012,第2期

机译：鉴定GCLC启动子中的新型抗氧化剂响应元素的年龄特异性NRF2结合：用于在老化大鼠肝脏中损失谷胱甘肽合成能力的补偿手段？
4. Evidence for a reduced transactivating capacity of CCAAT/enhancer binding protein (C/EBP) beta in Mycobacterium avium ssp. paratuberculosis (MAP) infected macrophages [C] . Sommer S, Jeckstadt S, Valentin-Weigand P, International Colloquium on Paratuberculosis . 2007

机译：减少CCAAT /增强子结合蛋白（C / EBP）β在分枝杆菌SSP中的转移能力降低的证据。 Paratuberculosis（地图）感染巨噬细胞
5. Positive and negative regulation of neuroendocrine gene expression by zinc finger transcription factors: Identification of a negative retinoic acid response element in the rat oxytocin promoter, and the cloning of a novel factor binding a conserved element critical for effective expression of the growth hormone gene [D] . Lipkin, Steven Monroe 1993

机译：锌指转录因子对神经内分泌基因表达的正向和负向调节：鉴定大鼠催产素启动子中的视黄酸阴性反应元件，并克隆与保守元件结合的新型因子，该元件对于生长激素基因的有效表达至关重要
6. Loss of the Nrf2 transcription factor causes a marked reduction in constitutive and inducible expression of the glutathione S-transferase Gsta1 Gsta2 Gstm1 Gstm2 Gstm3 and Gstm4 genes in the livers of male and female mice. [O] . Simon A Chanas, Qing Jiang, Michael McMahon, 2002

机译：Nrf2转录因子的缺失会导致雄性和雌性小鼠肝脏中谷胱甘肽S转移酶Gsta1Gsta2Gstm1Gstm2Gstm3和Gstm4基因的组成型和诱导型表达显着降低。
7. Identification of age-specific Nrf2 binding to a novel antioxidant response element locus in the Gclc promoter: a compensatory means for the loss of glutathione synthetic capacity in the aging rat liver? [O] . Swapna V. Shenvi, Eric Smith, Tory M. Hagen 2012

机译：鉴定GCLC启动子中的新型抗氧化剂响应元素的年龄特异性NRF2结合：用于衰老大鼠肝脏谷胱甘肽合成能力的补偿手段？
8. Identification of Potential Binding Agents for Densified Fuel Preparation from Municipal Solid Waste: Phase 1, Final Report [R] . Daugherty, K. E. 1986

机译：从城市固体废物中鉴定用于致密燃料制备的潜在结合剂：第1阶段，最终报告

Identification of age-specific Nrf2 binding to a novel ARE locus in the Gclc promoter: a compensatory means for the loss of glutathione synthetic capacity in the aging rat liver?

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