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Androgen Receptor Splice Variants Contribute to Prostate Cancer Aggressiveness through Induction of EMT and Expression of Stem Cell Marker Genes

机译：雄激素受体剪接变异体通过诱导EMT和干细胞标记基因的表达促进前列腺癌的侵略性

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BackgroundThe mechanism(s) by which androgen receptor (AR) splice variants contribute to castration-resistant prostate cancer (CRPC) is still lacking.

机译：背景技术仍然缺乏雄激素受体（AR）剪接变体促成去势抵抗性前列腺癌（CRPC）的机制。

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期刊名称 other
作者
Dejuan Kong; Seema Sethi; Yiwei Li; Wei Chen; Wael A. Sakr; Elisabeth Heath; Fazlul H. Sarkar;
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年(卷),期 -1(75),2
年度 -1
页码 161–174
总页数 24
原文格式 PDF
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中图分类
关键词
Androgen Receptor splice variants Cancer stem cells self-renewal 3 3′-diindolylmethane;

机译：雄激素受体剪接变体;癌症干细胞;自我更新;3;3-二吲哚甲烷;

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专利

1. Androgen Receptor Splice Variants Contribute to Prostate Cancer Aggressiveness Through Induction of EMT and Expression of Stem Cell Marker Genes [J] . Kong Dejuan, Sethi Seema, Li Yiwei, The Prostate . 2015,第2期

机译：雄激素受体剪接变体通过诱导EMT和干细胞标记基因的表达促进前列腺癌的侵略性
2. Androgen Receptor Splice Variants Activate Androgen Receptor Target Genes and Support Aberrant Prostate Cancer Cell Growth Independent of Canonical Androgen Receptor Nuclear Localization Signal [J] . Siu Chiu Chan, Yingming Li, Scott Dehm The Journal of biological chemistry . 2012,第23期

机译：雄激素受体剪接变体激活雄激素受体靶基因并支持异常前列腺癌细胞生长，与规范雄激素受体核定位信号无关
3. Melatonin Inhibits Androgen Receptor Splice Variant-7 (AR-V7)-Induced Nuclear Factor-Kappa B (NF-κB) Activation and NF-κB Activator-Induced AR-V7 Expression in Prostate Cancer Cells: Potential Implications for the Use of Melatonin in Castration-Resistant Prostate Cancer (CRPC) Therapy [J] . Vincent Wing Sun Liu, Wing Lung Yau, Chun Wai Tam, International Journal of Molecular Sciences . 2017,第6期

机译：褪黑素抑制前列腺癌细胞中雄激素受体剪接变异体7（AR-V7）诱导的核因子-κB（NF-κB）活化和NF-κB活化剂诱导的AR-V7表达：在体内使用褪黑激素的潜在影响去势抵抗性前列腺癌（CRPC）治疗
4. Role of the Androgen Receptor and PI3K/Akt in the Survival of Androgen-Refractory Prostate Cancer Cells [C] . Haojie Huang, Donald J. Tindal International Symposium on Hormonal Carcinogenesis . 2005

机译：雄激素受体和PI3K / AKT在雄激素难治性前列腺癌细胞存活中的作用
5. MED19 Alters Genomic Occupancy of the Androgen Receptor and Gene Expression in Prostate Cancer Cells, Driving Growth under Low Androgen [D] . Weber, Hannah. 2020

机译：Med19改变了前列腺癌细胞中雄激素受体和基因表达的基因组占据，在低雄激素下促进生长
6. Androgen Receptor Splice Variants Activate Androgen Receptor Target Genes and Support Aberrant Prostate Cancer Cell Growth Independent of Canonical Androgen Receptor Nuclear Localization Signal [O] . Siu Chiu Chan, Yingming Li, Scott M. Dehm 2012

机译：雄激素受体剪接变体激活雄激素受体靶基因并支持异常前列腺癌细胞的生长而与规范的雄激素受体核定位信号无关
7. Androgen receptor splice variants contribute to prostate cancer aggressiveness through induction of EMT and expression of stem cell marker genes [O] . Dejuan Kong, Seema Sethi, Yiwei Li, 2014

机译：雄激素受体剪接变体通过诱导EMT和干细胞标志物基因的表达导致前列腺癌侵略性有助于

Androgen Receptor Splice Variants Contribute to Prostate Cancer Aggressiveness through Induction of EMT and Expression of Stem Cell Marker Genes

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