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Endocytosis machinery is required for β1-adrenergic receptor-induced hypertrophy in neonatal rat cardiac myocytes

机译：β1肾上腺素能受体诱导的新生大鼠心肌细胞肥大需要胞吞作用机制

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摘要

AimsCardiac hypertrophy by activation of the β-adrenergic receptor (βAR) is mediated more efficiently by the β1-AR than by the β2-AR. We investigated the signalling mechanism by which the β1-AR mediates cardiac hypertrophy.

机译：通过β-肾上腺素能受体（βAR）的激活，心脏肥大由β1-AR比由β2-AR更有效地介导。我们研究了β1-AR介导心肌肥大的信号传导机制。

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期刊名称 Cardiovascular Research
作者
Carmine Morisco; Chiara Marrone; Jonathan Galeotti; Dan Shao; Dorothy E. Vatner; Stephen F. Vatner; Junichi Sadoshima;
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作者单位

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年(卷),期 -1(78),1
年度 -1
页码 36–44
总页数 9
原文格式 PDF
正文语种
中图分类心血管疾病;
关键词
Myocytes Cell culture Protein kinases Protein phosphorylation;

机译：心肌细胞;细胞培养;蛋白激酶;蛋白磷酸化;

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专利

1. Endocytosis machinery is required for beta1-adrenergic receptor-induced hypertrophy in neonatal rat cardiac myocytes. [J] . Morisco C, Marrone C, Galeotti J, Cardiovascular Research . 2008,第1期

机译：内吞作用机制是新生大鼠心肌细胞中β1-肾上腺素能受体诱导的肥大所必需的。
2. Endocytosis machinery is required for β₁-adrenergic receptor-induced hypertrophy in neonatal rat cardiac myocytes [J] . Carmine Morisco12 Chiara Marrone12 Jonathan Galeotti1 Dan Shao1 Dorothy E. Vatner1 Stephen F. Vatner1 and Junichi Sadoshima1* Cardiovascular Research . 2008,第1期

机译：β_{1 -肾上腺素能受体诱导的新生大鼠心肌细胞肥大需要胞吞作用机制}
3. Stimulation of the p38 Mitogen-activated Protein Kinase Pathway in Neonatal Rat Ventricular Myocytes by the G Protein–coupled Receptor Agonists, Endothelin-1 and Phenylephrine: A Role in Cardiac Myocyte Hypertrophy? [J] . Angela Clerk, Ashour Michael, Peter H. Sugden Journal of cell biology . 1998,第2期

机译：G蛋白偶联受体激动剂，内皮素-1和去氧肾上腺素对新生大鼠心室肌细胞中p38丝裂原活化蛋白激酶途径的刺激：在心肌肥大中的作用？
4. Simulations of Spontaneous Action Potentials via the Combination of β1-Adrenergic Stimulation and NCX Mutation in Cardiac Myocytes of Mouse Left Ventricle [C] . D.P.M. Esprito Santo, R.W. dos Santos, S.C. Lcilc Latin American Congress on Biomedical Engineering . 2015

机译：通过β1-肾上腺素能刺激和小鼠左心室心肌细胞的组合模拟β1-肾上腺素能刺激和NCX突变的组合
5. Cardiac stem cells generate new cardiac myocytes during normal cardiac development and pathological hypertrophy. [D] . Wilson, Rachel. 2008

机译：在正常心脏发育和病理性肥大过程中，心脏干细胞会产生新的心肌细胞。
6. The cardiac beta-myosin heavy chain isogene is induced selectively in alpha 1-adrenergic receptor-stimulated hypertrophy of cultured rat heart myocytes. [O] . L E Waspe, C P Ordahl, P C Simpson 1990

机译：心脏β-肌球蛋白重链同基因在培养的大鼠心脏心肌细胞的α1-肾上腺素受体刺激的肥大中被选择性诱导。
7. Endocytosis machinery is required for β1-adrenergic receptor-induced hypertrophy in neonatal rat cardiac myocytes [O] . Morisco, Carmine, Marrone, Chiara, Galeotti, Jonathan, 2008

机译：β1肾上腺素能受体诱导的新生大鼠心肌细胞肥大需要胞吞作用机制
8. Development of the Linkage of Beta-Adrenergic Receptors to Cardiac Hypertrophy and Heart Rate Control: Neonatal Sympathectomy with 6-Hydroxydopamine [R] . Hou, Q. C., Seidler, F. J., Slotkin, T. A. 1989

机译：β-肾上腺素能受体与心脏肥大和心率控制的联系的发展：新生儿交感神经切断术与6-羟基多巴胺

Endocytosis machinery is required for β1-adrenergic receptor-induced hypertrophy in neonatal rat cardiac myocytes

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