首页> 美国卫生研究院文献>Proceedings of the National Academy of Sciences of the United States of America >Correction for Jiang et al. Abnormal integrin-mediated regulation of chronic myelogenous leukemia CD34+ cell proliferation: BCR/ABL up-regulates the cyclin-dependent kinase inhibitor p27Kip which is relocated to the cell cytoplasm and incapable of regulating cdk2 activity

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Correction for Jiang et al. Abnormal integrin-mediated regulation of chronic myelogenous leukemia CD34+ cell proliferation: BCR/ABL up-regulates the cyclin-dependent kinase inhibitor p27Kip which is relocated to the cell cytoplasm and incapable of regulating cdk2 activity

机译：对Jiang等人的校正整联蛋白介导的慢性粒细胞性白血病CD34 +细胞增殖的异常调节：BCR / ABL上调细胞周期蛋白依赖性激酶抑制剂p27Kip后者重新定位到细胞质中且无法调节cdk2活性

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期刊名称 Proceedings of the National Academy of Sciences of the United States of America
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年(卷),期 2009(106),15
年度 2009
页码 6423
总页数 1
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1. Analysis of mutations in the BCR-ABL1 kinase domain, using direct sequencing: Detection of the T315I mutation in bone Marrow CD34+ cells of a patient with chronic myelogenous leukemia 6 months prior to its emergence in peripheral blood [J] . RazgaF., JurcekT., JeziskovaI., Molecular diagnosis & therapy . 2012,第3期

机译：使用直接测序分析BCR-ABL1激酶结构域中的突变：在外周血中出现6个月之前，对患有慢性粒细胞性白血病的患者的CD34 +细胞中的T315I突变进行检测
2. PHA-680626 exhibits anti-proliferative and pro-apoptotic activity on Imatinib-resistant chronic myeloid leukemia cell lines and primary CD34+ cells by inhibition of both Bcr-Abl tyrosine kinase and Aurora kinases. [J] . Gontarewicz A, Balabanov S, Keller G, Leukemia Research: A Forum for Studies on Leukemia and Normal Hemopoiesis . 2008,第12期

机译：PHA-680626通过抑制Bcr-Abl酪氨酸激酶和Aurora激酶，在对伊马替尼耐药的慢性粒细胞白血病细胞系和原代CD34 +细胞上具有抗增殖和促凋亡活性。
3. Efficacy and Safety of a Specific Inhibitor of the BCR-ABL Tyrosine Kinase in Chronic Myeloid Leukemia Activity of a Specific Inhibitor of the BCR-ABL Tyrosine Kinase in the Blast Crisis of Chronic Myeloid Leukemia and Acute Lympoblastic Leukemia wit [J] . B. J. Druker, M. Talpaz, D. J. Resta, Chemtracts . 2001,第13期

机译：BCR-ABL酪氨酸激酶特异性抑制剂在慢性粒细胞白血病中的功效和安全性BCR-ABL酪氨酸激酶特异性抑制剂在慢性粒细胞白血病和急性淋巴细胞白血病机智的爆发危机中的活性
4. Controlled Cellular Delivery of Bcr-Abl Coiled-Coil Domain for Apoptosis of Chronic Myelogenous Leukemia Cells [C] . Mudit Kakar, James R. Davis, Carol S. Lim Controlled Release Society Annual Meeting and Exposition . 2007

机译：慢性骨髓性白血病细胞凋亡的Bcr-Abl螺旋线圈结构域的受控细胞传递。
5. Regulation of Bcr-Abl signal transduction in the differentiation and survival of chronic myelogenous leukemia cells. [D] . Dorsey, Jay Fitzgerald. 2003

机译：Bcr-Abl信号转导在慢性粒细胞性白血病细胞分化和存活中的调控。
6. Abnormal integrin-mediated regulation of chronic myelogenous leukemia CD34+ cell proliferation: BCR/ABL up-regulates the cyclin-dependent kinase inhibitor p27Kip which is relocated to the cell cytoplasm and incapable of regulating cdk2 activity [O] . Yuehua Jiang, Robert C. H. Zhao, Catherine M. Verfaillie 2000

机译：整联蛋白介导的慢性粒细胞性白血病CD34 +细胞增殖的异常调节：BCR / ABL上调细胞周期蛋白依赖性激酶抑制剂p27Kip后者重新定位到细胞质中且无法调节cdk2活性
7. Correction for Jiang et al., Abnormal integrin-mediated regulation of chronic myelogenous leukemia CD34+ cell proliferation: BCR/ABL up-regulates the cyclin-dependent kinase inhibitor, p27Kip, which is relocated to the cell cytoplasm and incapable of regulating cdk2 activity [O] . 2009

机译：对Jiang等人的校正，整联蛋白介导的慢性粒细胞性白血病CD34 +细胞增殖的异常调节：BCR / ABL上调细胞周期蛋白依赖性激酶抑制剂p27Kip，后者重新定位到细胞质中且无法调节cdk2活性

Correction for Jiang et al. Abnormal integrin-mediated regulation of chronic myelogenous leukemia CD34+ cell proliferation: BCR/ABL up-regulates the cyclin-dependent kinase inhibitor p27Kip which is relocated to the cell cytoplasm and incapable of regulating cdk2 activity

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