首页> 美国卫生研究院文献>Medical Science Monitor : International Medical Journal of Experimental and Clinical Research >Acupuncture Administration Improves Cognitive Functions and Alleviates Inflammation and Nuclear Damage by Regulating Phosphatidylinositol 3 Kinase (PI3K)/Phosphoinositol-Dependent Kinase 1 (PDK1)/Novel Protein Kinase C (nPKC)/Rac 1 Signaling Pathway in Senescence-Accelerated Prone 8 (SAM-P8) Mice

【2h】

Acupuncture Administration Improves Cognitive Functions and Alleviates Inflammation and Nuclear Damage by Regulating Phosphatidylinositol 3 Kinase (PI3K)/Phosphoinositol-Dependent Kinase 1 (PDK1)/Novel Protein Kinase C (nPKC)/Rac 1 Signaling Pathway in Senescence-Accelerated Prone 8 (SAM-P8) Mice

机译：针灸管理通过调节磷脂酰肌醇3激酶（PI3K）/磷酸肌醇依赖性激酶1（PDK1）/新颖的蛋白激酶C（nPKC）/ Rac 1衰老加速倾向8（SAM-）信号通路来改善认知功能并减轻炎症和核损害。 P8）老鼠

代理获取

本网站仅为用户提供外文OA文献查询和代理获取服务，本网站没有原文。下单后我们将采用程序或人工为您竭诚获取高质量的原文，但由于OA文献来源多样且变更频繁，仍可能出现获取不到、文献不完整或与标题不符等情况，如果获取不到我们将提供退款服务。请知悉。

页面导航

摘要
著录项
相似文献
相关主题

摘要

BackgroundAlzheimer’s disease (AD) is an age-associated neurodegenerative disorder. This study aimed to investigate effects of acupuncture administration on cognitive function and associated mechanisms.

机译：背景阿尔茨海默氏病（AD）是一种与年龄相关的神经退行性疾病。本研究旨在研究针灸对认知功能及相关机制的影响。

著录项

期刊名称 Medical Science Monitor : International Medical Journal of Experimental and Clinical Research
作者
Guomin Li; Lirong Zeng; Haiyan Cheng; Jingxian Han; Xuezhu Zhang; Hui Xie;
展开▼
作者单位

展开▼
年(卷),期 2019(25),-1
年度 2019
页码 4082–4093
总页数 12
原文格式 PDF
正文语种
中图分类
关键词
Acupuncture Aging Alzheimer Disease Cognition;

机译：针灸;衰老;阿尔茨海默氏病;认知;

相似文献

外文文献
专利

1. Epicatechin induces NF- kappa B, activator protein-1 (AP-1) and nuclear transcription factor erythroid 2p45-related factor-2 (Nrf2) via phosphatidylinositol-3-kinase/protein kinase B (PI3K/AKT) and extracellular regulated kinase (ERK) signalling inHepG2 cells. [J] . Granado-Serrano A. B., Martin M. A., Haegeman G., The British Journal of Nutrition . 2010,第2期

机译：表儿茶素通过磷脂酰肌醇3激酶/蛋白激酶B（PI3K / AKT）和细胞外调节激酶（NF-κB，活化蛋白1（AP-1）和核转录因子红细胞2p45相关因子2（Nrf2）诱导。 ERK）在HepG2细胞中发出信号。
2. Epicatechin induces NF- kappaB, activator protein-1 (AP-1) and nuclear transcription factor erythroid 2p45-related factor-2 (Nrf2) via phosphatidylinositol-3-kinase/protein kinase B (PI3K/AKT) and extracellular regulated kinase (ERK) signalling inHepG2 cells. [J] . Granado-Serrano A. B., Martin M. A., Haegeman G., The British Journal of Nutrition . 2010,第2期

机译：表儿茶素通过磷脂酰肌醇3激酶/蛋白激酶B（PI3K / AKT）和细胞外调节激酶（ERK）诱导NF-κB，激活蛋白1（AP-1）和核转录因子红细胞2p45相关因子2（Nrf2）。）在HeepG2细胞中发出信号。
3. Epicatechin induces NF-κB, activator protein-1 (AP-1) and nuclear transcription factor erythroid 2p45-related factor-2 (Nrf2) via phosphatidylinositol-3-kinase/protein kinase B (PI3K/AKT) and extracellular regulated kinase (ERK) signalling in HepG2 cells [J] . Ana Belén Granado-Serrano, Guy Haegeman, Laura Bravo, The British Journal of Nutrition . 2009,第2期

机译：表儿茶素通过磷脂酰肌醇3激酶/蛋白激酶B（PI3K / AKT）和细胞外调节激酶（ERK）诱导NF-κB，激活蛋白1（AP-1）和核转录因子红细胞2p45相关因子2（Nrf2）。）在HepG2细胞中的信号转导
4. Role of the phosphatidylinositol (PI) 3-kinase/Akt protein kinase signaling pathway in the survival of rat sympathetic neurons [D] . Crowder, Robert J. 2000

机译：磷脂酰肌醇（PI）3-激酶/ Akt蛋白激酶信号通路在大鼠交感神经元存活中的作用
5. Identification of Functionally Distinct TRAF Proinflammatory and Phosphatidylinositol 3-Kinase/Mitogen-activated Protein Kinase/Extracellular Signal-regulated Kinase Kinase (PI3K/MEK) Transforming Activities Emanating from RET/PTC Fusion Oncoprotein [O] . Josephine H. F. Wixted, Jay L. Rothstein, Laurence C. Eisenlohr 2012

机译：RET / PTC融合癌蛋白产生的功能不同的TRAF促炎和磷脂酰肌醇3-激酶/丝裂原活化蛋白激酶/细胞外信号调节激酶激酶（PI3K / MEK）转化活性的鉴定
6. Acupuncture Administration Improves Cognitive Functions and Alleviates Inflammation and Nuclear Damage by Regulating Phosphatidylinositol 3 Kinase (PI3K)/Phosphoinositol-Dependent Kinase 1 (PDK1)/Novel Protein Kinase C (nPKC)/Rac 1 Signaling Pathway in Senescence-Accelerated Prone 8 (SAM-P8) Mice [O] . Guomin Li, Lirong Zeng, Haiyan Cheng, 2019

机译：针灸给药通过调节磷脂酰肌醇3激酶（PI3K）/磷酸肌醇依赖性激酶1（PDK1）/新蛋白激酶C（NPKC）/ RAC 1信号传导途径，通过调节磷脂酰肌醇3激酶（PI3K）/ Rac 1信号通路（SAM- p8）小鼠

Acupuncture Administration Improves Cognitive Functions and Alleviates Inflammation and Nuclear Damage by Regulating Phosphatidylinositol 3 Kinase (PI3K)/Phosphoinositol-Dependent Kinase 1 (PDK1)/Novel Protein Kinase C (nPKC)/Rac 1 Signaling Pathway in Senescence-Accelerated Prone 8 (SAM-P8) Mice

摘要

著录项

相似文献

相关主题

期刊订阅