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Information Biology on Low Intensity Laser

机译:低强度激光器的信息生物学

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In this paper, we showed that the photon-cell interaction of low intensity laser effects on cells in non-resonant. Although the non-resonant interaction itself is extraordinarily weak, it can be amplified by the coherent interaction. We then put forward the biological information model of low intensity laser (BIML) on cells and the biological information transformation model (BITML). According to BIML:laser couples with intracellular messenger through the chromophore absorption in the cell membrane: cold-color (green, blue, violet) activates cAMP phosphodiesterase through G_i protein or activates phosphoinositide phospholipase C through G_q protein, or activates one of receptor-linked enzyme: cAMP↓; hot-color (red, orange, yellow) activates adenylate cyclase through G_s protein: cAMP↑. According to BITML, if the laser irradiation dose is greater than the corresponding threshold value, its function would be the function of its complementary color under the non-damage condition. Their successful applications at the cellular level showed that BIML and BITML hold for laser-cell interaction.
机译:在本文中,我们表明,低强度激光效应对非共振细胞的光子 - 细胞相互作用。尽管非共振相互作用本身非常弱,但它可以通过相干的相互作用来放大。然后,我们提出了在细胞和生物信息转换模型(Bitml)上的低强度激光(BIML)的生物信息模型。根据BIML:激光与细胞内信使通过发色团吸收的细胞膜:冷颜色(绿色,蓝色,紫色)通过G_I蛋白激活CAMP磷酸二酯酶或通过G_Q蛋白激活磷酸膦酸磷脂酶C,或激活一组受体连接酶:营地↓;热颜色(红色,橙色,黄色)通过G_S蛋白激活腺苷酸环酶:营地↑。根据Bitml,如果激光照射剂量大于相应的阈值,则其功能将是其在非损伤条件下的互补颜色的功能。它们在蜂窝水平的成功应用表明,BIML和Bitml保持用于激光单元相互作用。

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