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Gigacycle fatigue: non-local and scaling aspects of damage localization, crack initiation and propagation

机译:缩放疲劳:非局部和缩放方面的损伤定位,裂纹启动和传播

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Progress in instrumentation in fatigue studies in the range 10~7 - 10~9 cycles revealed new paradigm: crack growth is not a main portion of life and initiation mechanisms are of primary importance with internal fatigue crack origin. Characteristic features of fatigue in this regime is strong evidence that Wohler-type S-N or Coffin-Manson diagrams can exhibit a second lower fatigue limit - multi-stage (duplex) fatigue life diagrams. An important finding in connection with these observations was that, in the transition from HCF to gigacycle regimes, the origins of fatigue failure changed from surface to interior "fish-eye" fracture. This experimental and theoretical study is focused on fatigue crack initiation and early slow crack growth as life-controlling factors of gigacycle fatigue. Nonlocal model of damage kinetics based on the description of collective behaviour of defects (slip bands, microshears, microcracks) allowed the interpretation of fatigue damage-failure transition as specific type of criticality in out-of-equilibrium system "solid with defects".
机译:在10〜7-10〜9循环范围内疲劳研究中的仪器进展揭示了新的范式:裂纹增长不是寿命的主要部分,并且引发机制具有初步的内部疲劳裂缝起源。这种制度疲劳的特征是强大的证据表明,Wohler型S-N或棺材 - 曼森图可以表现出第二次疲劳极限 - 多级(双工)疲劳寿命图。与这些观察结果相关的重要发现是,在从HCF转变为缩放制度,疲劳失效的起源从表面变为内部“鱼眼”骨折。这种实验和理论研究专注于疲劳裂纹引发和早期缓慢的裂纹增长,作为缩放疲劳的生命控制因素。基于缺陷的集体行为描述的非局部损伤动力学模型(滑动带,微裂缝,微裂纹)允许解释疲劳损伤失效过渡作为特定类型的均衡系统中的特定类型的关键性“坚实的缺陷”。

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