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Genetic Analysis on Fruit Cut Cracking of Watermelon

机译:西瓜切瓜开裂的遗传分析

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Inheritance of fruit cut cracking was analyzed by using the joint segregation analysis of mixed genetic models of major gene plus polygene in six generations (PI, P2, Fl, Bl, B2, andF2) derived from two crosses of L-103 (fruit-cracking susceptible) ×X-101 (fruitcracking resistance). The cut fruit cracking was dominated by two major genes with additive-dominance-epistatic effects plus polygenes with additive-dominance effects(E-l model) in Cross X-101 ×L-103. The hereditability of major genes (hmx2) were 40.34%,, 25.08%. 33.10% in Bl. B2, F2 population* and thai of polygenes (hpx2) detected in Bl. B2, F2population were 32.65%. 0. 50.20%. It also indicated that the environmental factors had a great effect on fruit cracking resistance as the environmental and genetic variance accounted for 39.55% and 60.45%, on average, of the phenotypic variance, respectively.
机译:通过对来自L-103的两个杂交(裂果)的六个世代(PI,P2,F1,B1,B2和F2)的主要基因加多基因混合遗传模型进行联合分离分析,分析了切果裂的遗传。 ×)X-101(耐果裂)。在X-101×L-103杂交中,切果裂由两个具有加性显性作用的主基因和具有加性显性作用的多基因(E-1模型)控制。主要基因(hmx2)的遗传力分别为40.34%,25.08%。 Bl的33.10%在Bl中检测到的B2,F2种群*和多基因(hpx2)的泰国人。 B2,F2人群为32.65%。 0. 50.20%。这也表明环境因素对水果抗裂性有很大影响,因为环境和遗传方差平均分别占表型方差的39.55%和60.45%。

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