首页> 中文会议>中国化学会全国第五届无机化学学术会议 >Bi<,2>Ln<,2>Ti<,2>MO<,12-y>(Ln=La、Nd、Gd、Dy;M=Ga、Al)的合成与电性质

Bi<,2>Ln<,2>Ti<,2>MO<,12-y>(Ln=La、Nd、Gd、Dy;M=Ga、Al)的合成与电性质

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《中国化学会全国第五届无机化学学术会议》|2000年|240-242|共3页
会议地点山东威海
作者
梁宏伟; 孟健; 王鸿燕; 彭程; 杜红初; 苏锵;
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作者单位

中国化学会;

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会议组织
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中图分类导电材料及其制品;电解与电极作用;
关键词
氧离子导体; 固体电解质; 电性质; 层状化合物;
入库时间 2022-08-17 10:25:37

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1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004）（P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
2. Ln(DMSO)7ClBPh42 (Ln=La、 Nd、 Sm Eu、 Gd、 Tb、 Dy、 Tm)的合成、性质及Eu(DMSO)7ClBPh42的晶体结构 [J] . 张道 ,郭松山 ,王汉章 . 无机化学学报 . 1999,第005期
3. LnAlOs中Ce3+，Tb3+，Dy3+的发光及Ce-Tb，Ce-Dy的敏化发光(Ln=La，Gd，Y) [J] . 王瑞萍 ,李有谟 . 发光学报 . 1990,第001期
4. NaLn4-x(SiO4)3F∶xRE3+(Ln=La,Gd；RE=Tb,Dy,Sm,Tm)荧光材料的发光性能 [J] . 郑成祥 ,梁利芳 ,郑金菊 . 发光学报 . 2013,第011期
5. LnnMConO3n＋1（Ln=Sm,Gd;M=Sr,Ba;n=1,2）的合成,结构,电和磁性质研究 [J] . 李思温 ,任玉芳 . 化学学报 . 1995,第011期
6. Ｌｎ<,n>ＭＣｏ<,n>Ｏ<,3n+1>（Ｌｎ＝Ｓｍ，Ｇｄ；Ｍ＝Ｓｒ，Ｂａ；ｎ＝１，２）的合成，结构，电性质和磁性研究 [C] . 李思温 . 中国稀土学会第三届学术年会 . 1994
7. Bi1-xLnxVO4(Ln=La,Sm,Nd,Gd,Eu,Dy,Y)的制备、表征及光催化分解水性质 [A] . 王期召 . 2009

Bi<,2>Ln<,2>Ti<,2>MO<,12-y>(Ln=La、Nd、Gd、Dy;M=Ga、Al)的合成与电性质

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