procedure for the separate extraction of ammonia and tar by distillation of coal with high content of constituents and for the manufacture of water gas or producer gas of coal.

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  机译： 用苏打水分离含二氧化碳的气体（尤其是煤蒸馏气体）中的氨和酸洗硫磺的方法

• 4. procedures for the extraction of tar, ammonia, and ammonia from the dry distillation gases [P] . 德国专利： DE000001032465A . 1958-06-19

  机译： 从干馏气体中提取焦油，氨和氨的程序

• 5. procedures for the extraction of valuable tar and gas from highly wasserhaltiger brown coal. [P] . AT92839B . 1923-05-25

  机译： 从高瓦斯哈尔蒂格褐煤中提取有价值的焦油和天然气的步骤。

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• 1. Development of a Raoult’s Law-Based Screening-Level Risk Assessment Methodology for Coal Tar and Its Application to Ten Tars Obtained from Former Manufactured Gas Plants in the Eastern United States [J] . Derick G. Brown . 环境保护（英文） . 2013,第6期
• 2. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 3. “我国高含油率炼油煤的资源状况和利用前景” 的研究煤焦油生产和加工利用技术的现状和动向（专题报告之四） [C] . . 中国煤炭、中国石油、中国地质学会青年科技联谊会 . 1998
• 4. 煤焦油粗蒽蒽含量和煤焦油萘含量的毛细管气相色谱检测方法研究 [A] . 王俊广 . 2007