The method of the mechanism of kappa alpha_and the method of the mechanism of kappa alpha_gas at the exit of kappa alpha_in the engine passing through the engine.

• 1. 基于伯努利定律的发动机尾气稀释降温装置及调试方法和调节方法 [P] . 中国专利： CN110159398A . 2019-08-23
• 2. 一种规模化生产伯氏生卡螨的方法 [P] . 中国专利： CN104488830B . 2017.07.18
• 3. The method and mechanism of the method and mechanism of the process and the mechanism of the p p p963kappa_alpha epsilon_mu_. [P] . GR32246B . 1967-06-03

  机译： pp p963的方法和机制进程的方法和机制pp p963 kappa_alpha epsilon_mu_。

• 4. Methods The protective mechanisms in EC and kappa___damage were studied. [P] . GR10524B . 1945-11-21

  机译： 方法研究了EC和κ损伤的保护机制。

• 5. The method and mechanism for the preparation of kappa alpha tau. [P] . GR15978B . 1955-01-25

  机译： 制备κατ的方法和机理。

• 1. Preface to the Special Issue on Atmospheric Oxidation Capacity, Ozone, and PM2.5 Pollution: Quantification Methods, Formation Mechanisms, Simulation, and Control [J] . Yuesi WANG, Zirui LIU 大气科学进展（英文版） . 2021,第007期
• 2. Study on the Missing Data Mechanisms and Imputation Methods [J] . Abdullah Z. Alruhaymi, Charles J. Kim 统计学期刊（英文） . 2021,第004期
• 3. Plasma hydrogenation a new method of reducing the kappa value of the low kappa polyimide film [C] . Yue Kuo, Taewoo Chung, Helinda Nominanda Materials Research Society Symposium . 2003
• 4. Mechanisms Underlying the Adverse Consequences of Stress: A Role for the Dynorphin/Kappa Opioid Receptor System, p38α MAPK, and the Serotonin Transporter [D] . Schindler, Abigail G. 2012
• 5. 2-mercaptoethanol restores the ability of nuclear factor kappa B (NF kappa B) to bind DNA in nuclear extracts from interleukin 1-treated cells incubated with pyrollidine dithiocarbamate (PDTC). Evidence for oxidation of glutathione in the mechanism of inhibition of NF kappa B by PDTC. [O] . P Brennan, L A ONeill 1996
• 6. Mechanism of inactivation of NF-kappa B by a viral homologue of I kappa b alpha. Signal-induced release of i kappa b alpha results in binding of the viral homologue to NF-kappa B. [O] . Tait, SW, Reid, EB, Greaves, DR, 2000

• 1. Prediction of Kappa Number in Eucalyptus Kraft Pulp Continuous Digester Using the Box amp;Jenkins Methodology [J] . Flávio M. Correia ,José V. Hallak d’Angelo ,Roger J. Zemp . 化学工程与科学期刊（英文） . 2014,第4期
• 2. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 3. Investigation of the Effects of Profile Shift in Helical Gear Mechanisms with Analytical and Numerical Methods [J] . Gultekin Karadere ,Ilhan Yilmaz . 力学国际期刊（英文） . 2018,第5期
• 4. AN-9B发动机意外停车机理分析及故障排除方法 [C] . 龚德先 . 中国航空学会第十二届全国直升机年会 . 1996
• 5. 希尔伯特—黄变换方法在GAT磁悬浮陀螺仪数据处理中的应用 [A] . 张爱能 . 2014