Method of the imitation of the enamels of limoges, comes and other on the body, such as oyogalith and plastics

• 1. 珐琅用玻璃材料、珐琅产品、珐琅产品的制造方法 [P] . 中国专利： CN112055700A . 2020-12-08
• 2. 裂纹珐琅釉浆、制备方法和裂纹珐琅釉面板的制备方法 [P] . 中国专利： CN110104951B . 2021.09.21
• 3. Method of the imitation of coloured enamel on a transparent plastic materials, which gives the illusion of the relief well as their surface, or even [P] . 法国专利： FR529363A . 1921-11-26

  机译： 在透明塑料材料上模仿有色搪瓷的方法，可以使浮雕以及其表面产生幻觉，甚至

• 4. New process for the production of the partitions in the imitation of the plastic stained glass former and enamels [P] . 法国专利： FR89393E . 1967-06-16

  机译： 模仿塑料彩色玻璃模型和搪瓷的隔板生产新工艺

• 5. The manufacture of plastic material of mosaics and of imitations of stained glass and enamels [P] . 法国专利： FR1421978A . 1965-12-17

  机译： 马赛克，仿制彩色玻璃和搪瓷塑料材料的制造

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• 5. METHODS FOR THE EVALUATION OF CREEP RELAXATION AND THE AMPLITUDE OF REVERSE PLASTIC STRAIN FOR BODIES SUBJECTED TO CYCLIC LOADING [C] . Haofeng Chen, Alan R.S. Ponter ASME(American Society of Mechanical Engineers) Pressure Vessels and Piping Conference: Design and Analysis Methods and Fitness for Service Evaluations for Pressure Vessels and Components; 20030720-20030724; Cleveland,OH; US . 2003
• 6. The curative bloom: An investigation of medicinal botanical imagery incorporating precious metals and enamel as body adornment [D] . Friedland, Mara 2009
• 7. A new method for estimating time since death by analysis of substances deposited on the surface of dental enamel in a body immersed in seawater [O] . Noboru Ishikawa, Yasuo Miake, Kei Kitamura, -1
• 8. Real-Time Whole-Body Imitation by Humanoid Robots and Task-Oriented Teleoperation Using an Analytical Mapping Method and Quantitative Evaluation [O] . Zhijun Zhang, Yaru Niu, Ziyi Yan, 2018
• 9. Method and apparatus for bonding a plastics sleeve onto a metallic body Patent [R] . King, C. B. 1967

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. 使用便携设备对两件清代掐丝珐琅器物珐琅釉的对比研究 [C] . 段鸿莺 ,高飞 ,亓昊楠 . 东亚文化遗产保护学会第六次国际学术研讨会 . 2017
• 3. 画珐琅工艺研究及画珐琅在首饰设计中的应用 [A] . 刘柳 . 2015