Timepiece movement which it is possible to add or remove instantaneously a striking mechanism of the hours and a striking mechanism of the quarters

• 1. 用于钟表机芯的发条盒轴、发条盒发条以及包括这种发条和/或这种轴的发条盒 [P] . 中国专利： CN104204965A . 2014-12-10
• 2. 发条盒的轴、发条及包括该轴和发条的发条盒、钟表机芯、腕表和表 [P] . 中国专利： CN108196438A . 2018-06-22
• 3. Timepiece movement which it is possible to add or remove instantaneously a striking mechanism of the hours and a striking mechanism of the quarters [P] . 法国专利： FR654118A . 1929-04-02

  机译： 钟表机芯，可以即时添加或删除小时的发条机制和四分之一的发条机制

• 4. Watch with mutually independent haen clock movement, hours -, half hours - and - striking mechanism quarter hours [P] . 德国专利： DE502153C . 1930-07-11

  机译： 带有相互独立的钟表机芯的手表，小时-半小时-和-撞击机构四分之一小时

• 5. Downstream stage for use in striking block of minute repeater striking mechanism controlled by horological movement of complicated watch, has hour ratchet that cooperates with hammer lifting piece of striking mechanism [P] . 瑞士专利： CH704623A2 . 2012-09-14

  机译： 下游级，用于由复杂表的钟表机芯控制的三问报时器发条机构中，具有小时棘轮，与锤发条机构配合

• 1. The Present-Day Crustal 3D Movement and Geodynamic Mechanisms in Hainan Island [J] . Yaxuan Hu, Jiuchang Hu, Bin Zhao, 地球科学和环境保护期刊（英文） . 2020,第005期
• 2. Using probabilistic movement primitives for striking movements [C] . Sebastian Gomez-Gonzalez, Gerhard Neumann, Bernhard Schölkopf, IEEE-RAS International Conference on Humanoid Robots . 2016
• 3. Characterization of Grease Interceptors for Removing Fat, Oil and Grease (FOG) and Mechanisms of FOG Deposit Formation in Sewer Systems. [D] . He, Xia. 2012
• 4. Striking similarities between the regulatory mechanisms governing yeast mating-type genes and mammalian major histocompatibility complex genes. [O] . J D Weissman, D S Singer 1991
• 5. Invoking freedom of expression and freedom of competition in trade mark infringement disputes: legal mechanisms for striking a balance [O] . Łukasz Żelechowski 2018

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Advances of Studies on Mechanisms of Drugs for Activating Blood Circulation and Removing Blood Stasis in Treatment of Primary Liver Cancer [J] . 任荣政 ,鞠立霞 ,陈喆 . 中医杂志：英文版 . 2005,第4期
• 3. An Eye-Movement Study on the Cognitive Neuroscience Mechanisms of Chinese Classifier Construction [J] . Yan Liu ,Junyan Wei ,Songsong Chen . 教育研究前沿：中英文版 . 2021,第001期
• 4. Identification of Bridge Movement Mechanisms [J] . Matthew T. Yarnold . 工程（英文）（1947-3931） . 2014,第9期
• 5. 探索建立“对破损档案即时发现即时提供抢救工作机制”的实践与思考 [C] . 张以群 . 纪念上海市档案馆建馆50周年学术研讨会暨第三届“‘3+1’档案论坛” . 2009
• 6. 中药材黄精种子萌发条件和休眠机制探究 [A] . 安瑞朋 . 2020