Method of processing of parts made of iron and steel, intended to protect them against the corrosive action of the water, air and other liquids or gases

• 1. 透析液体回路、具有透析液体回路的透析设备、流过透析液体回路的透析液体中的空气的检测方法以及气体传感器在透析液体回路中的应用 [P] . 中国专利： CN101687070B . 2013.02.27
• 2. 透析液体回路、具有透析液体回路的透析设备、流过透析液体回路的透析液体中的空气的检测方法以及气体传感器在透析液体回路中的应用 [P] . 中国专利： CN101687070A . 2010-03-31
• 3. A method for Treatment of Iron and Steel Structures to protect them against the corrosive Action of the water, Air and other liquids and gases. [P] . ES140637A1 . 1936-03-01

  机译： 一种钢铁结构的处理方法，可防止钢结构受到水，空气以及其他液体和气体的腐蚀作用。

• 4. Process for the protection of stainless steel against the action of corrosive liquids, highly acidic, as well as devices made of stainless steel protected by this process [P] . 法国专利： FR1097749A . 1955-07-08

  机译： 保护不锈钢免受强酸性腐蚀性液体影响的方法以及受此方法保护的不锈钢制成的设备

• 5. The process in order to protect the metal surfaces against the action of the air, water and of corrosive agents [P] . 法国专利： FR24762E . 1922-10-14

  机译： 为了保护金属表面免受空气，水和腐蚀剂作用的过程

• 1. Novel method for the determination of five carbamate pesticides in water samples by dispersive liquid-liquid microextraction combined with high performance liquid chromatography [J] . Zhi Mei Liu, Xiao Huan Zang, Wei Hua Liu, 中国化学快报（英文版） . 2009,第002期
• 2. Cleaning and Recycling of Industrial Wastes, Waste Waters and Off-Gases in the Iron and Steel Industry to Increase Environmetnal Protection [C] . Gerolf Strohmeier, Alfred E.Knieper, Rudoff Broeckl, International Symposium on Global environment and Iron and Steel Industry (ISES '98) April 14-16, 1998 Beijing, China . 1998
• 3. Study of fatigue process in air and corrosive environments using optical methods. [D] . Du, Ming Liang. 1998
• 4. Non-corrosive Green Lubricant With Dissolved Lignin in Ionic Liquids Behave as Ideal Lubricants for Steel-DLC Applications [O] . Jing Hua, Yijun Shi 2019
• 5. Effects of Polishing Methods on the Determination of Oxygen in Iron and Steel (Vacuum Fusion-Mass Spectrometric Analysis of Gases in Iron and Steel, 3rd Report) [O] . Ocirc, hiko Kammori, Naoharu Yamaguchi, 1968
• 6. Electron-Molecule Interactions in Gases and Liquids: The Electron-Attachment Process in Gases and Nonpolar Liquids [R] . Christophorou, L. G., Siomos, K. 1983

• 1. High Cr white cast iron/carbon steel bimetal liner by lost foam casting with liquid-liquid composite process [J] . Xiao Xiaofeng ,Ye Shengping ,Yin Weixin . 中国铸造 . 2012,第002期
• 2. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 3. Optimizing Sampling, Sample Processing and Analysis Methods for Radon (SUP222/SUPRn) in Water by Liquid Scintillation Counting [J] . Uttam K. Saha ,Michael E. Kitto ,Dana R. Lynch . 美国分析化学（英文） . 2018,第1期
• 4. Effect of welding process methods on the regularity of iron element in copper/steel deposited layer [J] . Wang Yan ,Li Longqing ,Lu Hong . 中国焊接 . 2013,第002期
• 5. 防止45钢零件开裂的方法 [C] . 施家山 ,叶利毅 ,施文静 . 第十八届华东六省一市热处理年会 . 2012