A method for the separate representation of the thyrotropic and gonadotropic hormone of the hypophyses front flap

• 1. 一种向半滑舌鳎垂体细胞添加维生素 E 后提高促性腺激素表达水平的方法 [P] . 中国专利： CN105838663B . 2019.11.26
• 2. 一种向半滑舌鳎垂体细胞添加维生素 E 后提高促性腺激素表达水平的方法 [P] . 中国专利： CN105838663A . 2016-08-10
• 3. A method for the separate production of a substance acting on the gonadotropic hypophyses front and of the tabs hormone from the hypophyses front tabs [P] . 德国专利： DE722571C . 1942-07-14

  机译： 一种单独生产作用于促性腺垂体前叶的物质和垂体前叶的垂体激素的生产方法

• 4. A process for the recovery of the purified gonadotropic hormones of the hypophyses front flap [P] . 德国专利： DE650803C . 1937-10-01

  机译： 纯化垂体前皮瓣的促性腺激素的过程

• 5. A method for the separate production of hypophyses behind tabs hormone and hypophyses front tabs hormone [P] . 德国专利： DE559143C . 1932-09-16

  机译： 一种单独产生垂体后部激素和垂体前体激素的方法

• 1. Uncertainty Representation of Ocean Fronts Based on Fuzzy-Rough Set Theory [J] . XUE Cunjin, ZHOU Chenghu, SU Fenzhen, 中国海洋大学学报（英文版） . 2008,第002期
• 2. Double J Fixation after Craniotomy: Technical Description of a Modification Method for Bone Flap Fixation (Hiederov Method) [J] . Hieder Al-Shami, Ahmed M. Salah, Mohamed Fathy Adel Ali 现代神经外科学进展（英文） . 2020,第003期
• 3. Clinical Report: Gonadotropic Releasing Hormone Agonist Implants [C] . Kimberly Cook, Gary Riggs Annual Conference of the Association of Avian Veterinarians . 2007
• 4. A Review of Genetic Polymorphisms in the Receptors for Gonadotropic and Sex Hormones in Polycystic Ovary Syndrome [D] . Rudolph, Sara 2017
• 5. Ineffectiveness in birds of antisera for mammalian gonadotropic and thyrotropic substances [O] . A. S. Parkes, I. W. Rowlands 1937
• 6. Ineffectiveness in birds of antisera for mammalian gonadotropic and thyrotropic substances [O] . Parkes, A. S., Rowlands, I. W. 1937
• 7. FRACTIONATION OF RAT PITUITARIES AND STUDY OF THE THYROTROPIC, GONADOTROPIC AND HETEROTHYROTROPIC ACTTVITIES OF THE PURIFIED PRODUCTS [R] . Y. A. Fontaine, E. Burzawa-Gerard 1970

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Repairing small wounds around ankle by medial planta island flaps pedicled with anterior tibial artery perforator in front of inner malleolus [J] . 魏在荣 . 外科研究与新技术 . 2011,第4期
• 3. Comparison of shape representation methods for dynamic cell analysis [J] . 李衡 ,刘志文 ,安兴 . 北京理工大学学报：英文版 . 2014,第4期
• 4. 人绒毛膜促性腺激素β亚基(hCGβ)和绵羊垂体促性腺激素共有α亚基(oLHα)嵌合肽基因在巴斯德毕赤酵母中的表达 [C] . 申庆祥 ,王健 ,沈卫英 . 第五届全国生殖免疫学大会 . 2001
• 5. 垂体促甲状腺激素分泌瘤6例分析与文献复习 [A] . 徐桦敏 . 2021