A method making it possible to increase the prolongation of the plastic resin linear irradiated

• 1. 一种低色热塑性高分子量线性饱和聚酯树脂的制备方法 [P] . 中国专利： CN112250844A . 2021-01-22
• 2. 一种高韧性高断裂伸长率长玻纤增强热塑性材料及其制备方法 [P] . 中国专利： CN109679221A . 2019-04-26
• 3. PROCESS FOR INCREASING THE ELONGATION OF IRRADIATED LINEAR RESINOUS PLASTIC MATERIALS [P] . CA591595A . 1960-01-26

  机译： 增加辐照线性树脂材料伸长率的方法

• 4. Process for increasing the elongation of irradiated linear resinous plastic materials [P] . 英国专利： GB855874A . 1960-12-07

  机译： 增加辐照线性树脂塑料材料的伸长率的方法

• 5. A method for synthetic resin coating of surfaces and plastic resin mixtures on the basis of this by means of unsaturated polyesters modified epoxy resins in this way an aet by irradiation are curable [P] . 德国专利： DE2003818A1 . 1970-08-06

  机译： 以此方式借助于不饱和聚酯改性的环氧树脂合成表面和塑料树脂混合物的合成树脂的方法是可固化的。

• 1. THE VIRTUAL WORK PRINCIPLE AND LINEAR COMPLEMEN-TARY METHOD FOR COUPLING ANALYSIS OF ELASTO-PLASTIC DAMAGE STRUCTURE [J] . 马景槐 应用数学和力学：英文版 . 2000,第002期
• 2. A CONDENSED METHOD FOR LINEAR COMPLEMENTARY EQUATION OF ELASTO－PLASTIC PROBLEMS [J] . 殷福新, 孙焕纯 应用数学和力学：英文版 . 1995,第005期
• 3. A CONDENSED METHOD FOR LINEAR COMPLEMENTARY EQUATIONS OF ELASTO－PLASTIC PROBLEMS [J] . 殷福新, 孙焕纯 应用数学和力学：英文版 . 1995,第010期
• 4. Establishment and validation of a method for cell irradiation in 96-well and 6-well plates using a linear accelerator [J] . Xiao-Qing Dong, Qing Lin, Jie Hu, 核技术（英文版） . 2018,第005期
• 5. Comparison of the bond strength of composite resin to Er:YAG laser-irradiated enamel/dentin pre-treated with various methods [C] . Toru Eguro, Toru Maeda, Yaeko Ishizak, Nihon Rēzā Shigakkai . 2003
• 6. Comparing material impact strength of three high impact plastic resins used in automotive grille using nonlinear finite element analysis tools. [D] . Santiago, Jason. 2005
• 7. The apparent increase in the β-parameter of the linear quadratic model with increased linear energy transfer during fast neutron irradiation [O] . B Jones 2010
• 8. Evaluation of Epoxy Resin for Cryogenic Use by Positron Annihilation Method. Change of Characteristics of Epoxy Resin with Plasticizer and Positron Lifetime. [O] . Shigehiro NISHIJIMA, Yoshihide HONDA, Yoshinori KOBAYASHI, 1994
• 9. Development of Laminating Resins, Controlled Processes, and Test Methods for Glass Filament Wound Reinforced Plastic [R] . 1939

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. 一种高性能热塑性树脂基复合材料及其高温力学性能 [C] . LIU Xinyu ,刘新宇 ,WANG Jinyan . 第一届中国国际复合材料科技大会 . 2013
• 3. 热塑性聚酯/热固性树脂共混方法制备环氧树脂微球研究 [A] . 吴洋 . 2008