method to increase the anreicherungsfaktors heavy water in the catalytic kohlenoxydhydrierung reaktionswasser

• 1. 造粒装置、制备四氯化硅催化氢化反应用催化剂的制备方法及四氯化硅催化氢化反应方法 [P] . 中国专利： CN105170022B . 2017.11.10
• 2. 造粒装置、制备四氯化硅催化氢化反应用催化剂的制备方法及四氯化硅催化氢化反应方法 [P] . 中国专利： CN105170022A . 2015-12-23
• 3. a method of increasing the anreicherungsfaktors heavy water to values above 1.1, especially about 1.35 [P] . AT208816B . 1960-04-25

  机译： 一种增加anreicherungsfaktors重水至1.1以上，尤其是1.35以上的值的方法

• 4. method to increase the yield of oxygenated products in the mitteldrucksynthese of gasoline by catalytic kohlenoxydhydrierung [P] . 德国专利： DE000000907050B . 1954-03-22

  机译： 催化三苯氧基水合提高汽油二甲醚合成中氧化产物收率的方法

• 5. method for increasing the kornfestigkeit of iron containing fällungskatalysatoren for catalytic kohlenoxydhydrierung [P] . AT175557B . 1953-07-25

  机译： 催化铁代酚醛加氢剂中铁的含量增加的方法

• 1. Determination of Trace Vanadium（V）in Seawater and Fresh Water by the Catalytic Kinetic Spectrophotometric Method [J] . JI Hongwei, SHA Yuanyuan, XIN Huizhen, 中国海洋大学学报：英文版 . 2010,第004期
• 2. Determination of Trace Vanadium(V)in Seawater and Fresh Water by the Catalytic Kinetic Spectrophotometric Method [J] . JI Hongwei, SHA Yuanyuan, XIN Huizhen, 中国海洋大学学报（英文版） . 2010,第004期
• 3. Assessment of Different Instruments and Methods for Detecting Pollution Share Rate of Heavy Metals in Water Samples [J] . Wen CHEN, Xiangjun WANG, Yang ZHAO, 农业生物技术：英文版 . 2014,第002期
• 4. Simultaneous Determination of Iron and Manganese in Water Using Artificial Neural Network Catalytic Spectrophotometric Method [J] . JI Hongwei, XU Yan, LI Shuang, 中国海洋大学学报（英文版） . 2012,第003期
• 5. HIERARCHICAL ZEOLITES: INCREASE IN MESOSURFACE VIA “BOTTOM-UP” OR “TOP- DOWN” METHODS AND ITS INFLUENCE IN CATALYTIC CRACKING [C] . Eduardo Falabella Sousa-Aguiar, Eledir Vitor Sobrinho, Alexandre Figueiredo Costa American Chemical Society National Meeting Exposition . 2015
• 6. Alternative Methods of Microalgae Cultivation and Dewatering to Increase the Sustainability of Biodiesel Production. [D] . Kring, Stefanie A. 2014
• 7. Heavy Sugar and Heavy Water Create Tunable Intact Protein Mass Increases for Quantitative MS in any Feed and Organism [O] . Jeniffer V. Quijada, Nicholas D. Schmitt, Joseph P. Salisbury, -1
• 8. Simulation Procedure for Multistage Water/Hydrogen Exchange Column for Heavy Water Enrichment Accounting for Catalytic and Scrubbing Efficiencies [O] . Masahiro KINOSHITA 1985
• 9. Structure-Sensitive Catalytic Dehydrogenation of Hydrogen Donors over Iron Sulfides: New Methods for Increasing Coal Depolymerization under Mild Conditions. Final Report, July 1, 1983-December 31, 1984 [R] . Smith, G. V., Phillips, J. B. 1985

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. 重水研究堆重水中阴离子和草酸根 [C] . 朱宝珍 . 第六届全国离子色谱学术报告会 . 1997
• 3. 铁催化二氧化碳和甲酸酯衍生物的氢化反应研究 [A] . 诸葛玲 . 2016