Process for the increase in the ability of paper to be corrugated, thanks to the use of residual lyes modified paste

• 1. 通过破坏gntR而提高了生产能力的异丙醇生产细菌 [P] . 中国专利： CN103068968B . 2014.07.23
• 2. 通过破坏gntR而提高了生产能力的异丙醇生产细菌 [P] . 中国专利： CN103068968A . 2013-04-24
• 3. Method of processing of the residual lyes of paper pulp [P] . 法国专利： FR1444499A . 1966-07-01

  机译： 纸浆残余碱液的处理方法

• 4. The process in order to increase the resistance to bad weather of agglomerated by means of the residual lyes of the cellulose sulphite [P] . 法国专利： FR415566A . 1910-09-29

  机译： 为了通过亚硫酸纤维素的残留碱液来提高抗结块性的方法

• 5. Process for the recovery of chemical products from the residual lyes from the manufacture of paper pulp [P] . 法国专利： FR1351412A . 1964-02-07

  机译： 从纸浆生产中的残留碱液中回收化学产品的方法

• 1. A Study of the Ability of Modified Dyeing, and Post-Dyeing Processes to Increase the Crystallinity of Dyestuffs and the Lightfastness of Wool Textiles [C] . Steven J. McNeil, Yi Hu, Larissa I. Zaitseva International Wool Research Conference . 2016
• 2. Reconstitution of dewatered food processing residuals with manure to increase energy production from anaerobic digestion. [D] . Wall, David M. 2010
• 3. Testing day: The effects of processing bias induced by Navon stimulion the strength of the Müller-Lyer illusion [O] . Matthew E. Mundy 2014
• 4. Adsorption-Photometric and Test-Determination of Fe(III) in Natural Water Using Silica Lyer-by-Lyer Modified with Polyhexamethylene Guanidine and Ferron [O] . Svetlana L. Didukh-Shadrina, Olga V. Buyko, Vladimir N. Losev 2020
• 5. Water Pollution Abatement by Improved Coagulation on Effluents from Lye-Peel Potato Processing Plants [R] . Fossum, G. O., Cooley, A. M. 1970

• 1. Jiangsu Shangshan Paper 200,000 t/a High Strength Corrugating Medium Paper Production Line Put into Operation [J] . . 造纸信息 . 2017,第8期
• 2. Jicheng Paper 102,000 t/a High Strength Corrugating Medium Paper Project Completed [J] . . 造纸信息 . 2016,第8期
• 3. Increasing Pupil Size Is Associated with Increasing Cognitive Processing Demands: A Pilot Study Using a Mobile Eye-Tracking Device [J] . Adam Szulewski ,Shannon M. Fernando ,Jared Baylis . 急诊医学（英文） . 2014,第1期
• 4. Increased Visual Acuity Will Not Necessarily Equal an Increased Reading Ability in Patients with Subfoveal Neovascular Macular Degeneration [J] . Fredrik P.Kallmark ,Anders Kvanta ,Gustaf Oqvist . 临床医学国际期刊（英文） . 2011,第4期
• 5. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 6. 乳酸脱氢酶2基因的缺失提高了苏云金芽胞杆菌的碱耐受性 [C] . 考桂伟 ,邱丽丽 ,彭琦 . 全国农业生物化学与分子生物学第十四届学术研讨会 . 2015
• 7. 废旧瓦楞纸回用过程中纤维物理性能变化及改性研究 [A] . 巫山 . 2011